WordNet
- (of structures or institutions) having lost stability; failing or on the point of collapse; "a tottering empire"
- unsteady in gait as from infirmity or old age; "a tottering skeleton of a horse"; "a tottery old man" (同)tottery
English Journal
- Absence-like seizures and their pharmacological profile in tottering-6j mice.
- Kim TY1, Maki T2, Zhou Y3, Sakai K2, Mizuno Y2, Ishikawa A2, Tanaka R2, Niimi K1, Li W3, Nagano N2, Takahashi E4.
- Biochemical and biophysical research communications.Biochem Biophys Res Commun.2015 Jul 17;463(1-2):148-53. doi: 10.1016/j.bbrc.2015.05.050. Epub 2015 May 20.
- We previously showed that recessive ataxic tottering-6j mice carried a base substitution (C-to-A) in the consensus splice acceptor sequence linked to exon 5 of the α1 subunit of the Cav2.1 channel gene (Cacna1a), resulting in the skipping of exon 5 and deletion of part of the S4-S5 linker, S5, and
- PMID 26002462
- Cerebellar output controls generalized spike-and-wave discharge occurrence.
- Kros L1, Eelkman Rooda OH1, Spanke JK1, Alva P2, van Dongen MN3, Karapatis A3, Tolner EA4, Strydis C1, Davey N2, Winkelman BH5, Negrello M1, Serdijn WA3, Steuber V2, van den Maagdenberg AM4,6, De Zeeuw CI1,5, Hoebeek FE1.
- Annals of neurology.Ann Neurol.2015 Jun;77(6):1027-49. doi: 10.1002/ana.24399.
- OBJECTIVE: Disrupting thalamocortical activity patterns has proven to be a promising approach to stop generalized spike-and-wave discharges (GSWDs) characteristic of absence seizures. Here, we investigated to what extent modulation of neuronal firing in cerebellar nuclei (CN), which are anatomically
- PMID 25762286
- Role of Synchronous Activation of Cerebellar Purkinje Cell Ensembles in Multi-joint Movement Control.
- Hoogland TM1, De Gruijl JR2, Witter L2, Canto CB2, De Zeeuw CI3.
- Current biology : CB.Curr Biol.2015 May 4;25(9):1157-65. doi: 10.1016/j.cub.2015.03.009. Epub 2015 Apr 2.
- It is a longstanding question in neuroscience how elaborate multi-joint movements are coordinated coherently. Microzones of cerebellar Purkinje cells (PCs) are thought to mediate this coordination by controlling the timing of particular motor domains. However, it remains to be elucidated to what ext
- PMID 25843032
- Abnormal excitability and episodic low-frequency oscillations in the cerebral cortex of the tottering mouse.
- Cramer SW1, Popa LS1, Carter RE1, Chen G1, Ebner TJ2.
- The Journal of neuroscience : the official journal of the Society for Neuroscience.J Neurosci.2015 Apr 8;35(14):5664-79. doi: 10.1523/JNEUROSCI.3107-14.2015.
- The Ca(2+) channelopathies caused by mutations of the CACNA1A gene that encodes the pore-forming subunit of the human Cav2.1 (P/Q-type) voltage-gated Ca(2+) channel include episodic ataxia type 2 (EA2). Although, in EA2 the emphasis has been on cerebellar dysfunction, patients also exhibit episodic,
- PMID 25855180
Japanese Journal
- 頸性めまい症例における重心動揺検査の特徴的検査所見の検討
- 田浦 晶子,清水 章子,細見 佳子,鳥居 紘子,荻野 枝里子,扇田 秀章,船曳 和雄,伊藤 壽一
- Equilibrium research 71(2), 87-95, 2012-04-01
- … From an analysis of stabilometry, the control group exhibited some tottering only in the head-extended position. … Most of the patients with cervical vertigo, however, readily displayed tottering in any neck position. …
- NAID 10030614629
- Striking pattern of Purkinje cell loss in cerebellum of an ataxic mutant mouse, tottering
- Sawada Kazuhiko,Azad Abul Kalam,Sakata-Haga Hiromi,Lee Nam-Seob,Jeong Young-Gil,Fukui Yoshihiro
- ACTA NEUROBIOLOGIAE EXPERIMENTALIS 69(1), 138-145, 2009
- NAID 120004710501
- Expression of tyrosine hydroxylase in cerebellar Purkinje cells of ataxic mutant mice:its relation to the onset and/or development of ataxia
- Sawada Kazuhiko,Fukui Yoshihiro,澤田 和彦,福井 義浩
- The journal of medical investigation : JMI 48(1/2), 5-10, 2001
- … An increased expression of TH in some Purkinje cells has been observed in two allelic groups of mutant mice, tottering and dilute. …
- NAID 110001900425
- Ataxic mutant mice with defects in Ca^<2+> channel a_<1A> subunit gene : morphological and functional abnormalities in cerebellar cortical neurons
- Sawada Kazuhiko,Haga Hiromi,Fukui Yoshihiro
- Congenital anomalies 40(2), 99-107, 2000-06
- NAID 110002785623
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