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1. 難治性狭心症マネージメントのための血管新生療法 therapeutic angiogenesis for management of refractory angina
2. 冠動脈内皮細胞機能不全：基本概念 coronary artery endothelial dysfunction basic concepts
3. AIDS関連リンパ腫：疫学、危険因子、および病理学 aids related lymphomas epidemiology risk factors and pathobiology
4. 肝線維症の病因 pathogenesis of hepatic fibrosis
5. BおよびTリンパ球の正常な発生 normal b and t lymphocyte development

English Journal

Coronin-1C Protein and Caveolin Protein Provide Constitutive and Inducible Mechanisms of Rac1 Protein Trafficking.

Williamson RC1, Cowell CA1, Reville T1, Roper JA1, Rendall TC2, Bass MD3.
The Journal of biological chemistry.J Biol Chem.2015 Jun 19;290(25):15437-49. doi: 10.1074/jbc.M115.640367. Epub 2015 Apr 29.
Sustained directional fibroblast migration requires both polarized activation of the protrusive signal, Rac1, and redistribution of inactive Rac1 from the rear of the cell so that it can be redistributed or degraded. In this work, we determine how alternative endocytic mechanisms dictate the fate of
PMID 25925950

Anesthetic propofol overdose causes vascular hyperpermeability by reducing endothelial glycocalyx and ATP production.

Lin MC1,2, Lin CF3, Li CF4, Sun DP5, Wang LY6, Hsing CH7,8.
International journal of molecular sciences.Int J Mol Sci.2015 May 27;16(6):12092-107. doi: 10.3390/ijms160612092.
Prolonged treatment with a large dose of propofol may cause diffuse cellular cytotoxicity; however, the detailed underlying mechanism remains unclear, particularly in vascular endothelial cells. Previous studies showed that a propofol overdose induces endothelial injury and vascular barrier dysfunct
PMID 26023717
PMID 25995222

Molecular mechanisms of syndecan-4 upregulation by TNF-α in the endothelium-like EAhy926 cells

Changes in proliferation, differentiation, fibroblast growth factor 2 responsiveness and expression of syndecan-4 and glypican-1 with turkey satellite cell age