WordNet

involving or derived from the senses; "sensory experience"; "sensory channels" (同)sensorial

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1. 感覚消失患者へのアプローチ approach to the patient with sensory loss
2. 遺伝性感覚自律神経ニューロパチー hereditary sensory and autonomic neuropathies
3. 成人におけるギラン・バレー症候群の臨床的特徴および診断 clinical features and diagnosis of guillain barre syndrome in adults
4. 免疫誘導性神経障害 immune mediated neuropathies
5. 神経伝導検査の概要 overview of nerve conduction studies

The influence of visual information on auditory processing in individuals with congenital amusia: An ERP study.

Lu X1, Ho HT2, Sun Y3, Johnson BW3, Thompson WF4.
NeuroImage.Neuroimage.2016 Apr 27;135:142-151. doi: 10.1016/j.neuroimage.2016.04.043. [Epub ahead of print]
While most normal hearing individuals can readily use prosodic information in spoken language to interpret the moods and feelings of conversational partners, people with congenital amusia report that they often rely more on facial expressions and gestures, a strategy that may compensate for deficits
PMID 27132045

Leveque Y1, Fauvel B, Groussard M, Caclin A, Albouy P, Platel H, Tillmann B.
Journal of neurophysiology.J Neurophysiol.2016 Mar 23:jn.00663.2015. doi: 10.1152/jn.00663.2015. [Epub ahead of print]
Congenital amusia, a neuro-developmental disorder of music perception and production, has been associated to abnormal anatomical and functional connectivity in a right fronto-temporal pathway. To investigate whether spontaneous connectivity in brain networks involving the auditory cortex is altered
PMID 27009161

Impaired encoding of rapid pitch information underlies perception and memory deficits in congenital amusia.

Albouy P1,2,3,4, Cousineau M3, Caclin A1,2, Tillmann B1,2, Peretz I3.
Scientific reports.Sci Rep.2016 Jan 6;6:18861. doi: 10.1038/srep18861.
Recent theories suggest that the basis of neurodevelopmental auditory disorders such as dyslexia or specific language impairment might be a low-level sensory dysfunction. In the present study we test this hypothesis in congenital amusia, a neurodevelopmental disorder characterized by severe deficits
PMID 26732511