WordNet
- a cellular structure that is postulated to exist in order to mediate between a chemical agent that acts on nervous tissue and the physiological response
- the secretion of an endocrine gland that is transmitted by the blood to the tissue on which it has a specific effect (同)endocrine, internal_secretion
- of or involving the pancreas; "pancreatic cancer"
PrepTutorEJDIC
- =sense organ / 受信装置
- ホルモン
- 膵臓の
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- 1. 転移性膵神経内分泌腫瘍および低分化胃腸膵管神経内分泌腫瘍：腫瘍増殖をコントローするための全身療法およびホルモン分泌過剰の症状 metastatic pancreatic neuroendocrine tumors and poorly differentiated gastroenteropancreatic neuroendocrine carcinomas systemic therapy options to control tumor growth and symptoms of hormone hypersecretion
- 2. 膵臓ステント留置術およびその合併症の概要 overview of pancreatic stenting and its complications
- 3. 膵神経内分泌腫瘍（膵島細胞腫瘍）の分類、疫学、臨床症状、局在、および病期分類 classification epidemiology clinical presentation localization and staging of pancreatic neuroendocrine tumors islet cell tumors
- 4. 甲状腺ホルモンの合成および生理 thyroid hormone synthesis and physiology
- 5. 膵外分泌癌の治療における外科手術の概要 overview of surgery in the treatment of exocrine pancreatic cancer and prognosis
English Journal
- PPARγ-inactive Δ2-troglitazone independently triggers ER stress and apoptosis in breast cancer cells.
- Colin-Cassin C1, Yao X, Cerella C, Chbicheb S, Kuntz S, Mazerbourg S, Boisbrun M, Chapleur Y, Diederich M, Flament S, Grillier-Vuissoz I.
- Molecular carcinogenesis.Mol Carcinog.2015 May;54(5):393-404. doi: 10.1002/mc.22109. Epub 2013 Nov 30.
- Our aim was to better understand peroxisome proliferator-activated receptor gamma (PPARγ)-independent pathways involved in anti-cancer effects of thiazolidinediones (TZDs). We focused on Δ2-troglitazone (Δ2-TGZ), a PPARγ inactive TZD that affects breast cancer cell viability. Appearance of TUNEL
- PMID 24293218
- Abscisic Acid transport in Human Erythrocytes.
- Vigliarolo T1, Guida L2, Millo E2, Fresia C2, Turco E3, De Flora A2, Zocchi E2.
- The Journal of biological chemistry.J Biol Chem.2015 Apr 6. pii: jbc.M114.629501. [Epub ahead of print]
- Abscisic acid (ABA) is a plant hormone involved in the response to environmental stress. Recently, ABA has been shown to be present and active also in mammals, where it stimulates the functional activity of innate immune cells, of mesenchymal and hemopoietic stem cells and of insulin-releasing pancr
- PMID 25847240
- Loss of fibroblast growth factor 21 action induces insulin resistance, pancreatic islet hyperplasia and dysfunction in mice.
- So WY1, Cheng Q1, Xu A2, Lam KS2, Leung PS1.
- Cell death & disease.Cell Death Dis.2015 Mar 26;6:e1707. doi: 10.1038/cddis.2015.80.
- Fibroblast growth factor (FGF) 21 is an endocrine factor that normalizes glucose homeostasis and reduces insulin resistance in diabetes. Although the pancreas is an FGF21 target organ, its role in pancreatic islets remains obscure. This study aimed to elucidate the physiological role of FGF21 in pan
- PMID 25811804
Japanese Journal
- Induction of thyroid remission using rituximab in a patient with type 3 autoimmune polyglandular syndrome including Graves' disease and type 1 diabetes mellitus: A case report
- , , , , , ,
- Endocrine Journal 62(1), 69-75, 2015
- … To protect pancreatic β cells, we administered RTX at a dose of 500 mg (approximately 300 mg/m2) on 2 occasions 1 week apart. … After treatment, no adverse effects were observed, and thyroid stimulating hormone receptor antibody (TRAb) was no longer detectable 4 months after RTX administration. …
- NAID 130004770470
- Induction of thyroid remission using rituximab in a patient with type 3 autoimmune polyglandular syndrome including Graves' disease and type 1 diabetes mellitus: A case report
- , , , , , ,
- Endocrine Journal, 2014
- … To protect pancreatic β cells, we administered RTX at a dose of 500 mg (approximately 300 mg/m2) on 2 occasions 1 week apart. … After treatment, no adverse effects were observed, and thyroid stimulating hormone receptor antibody (TRAb) was no longer detectable 4 months after RTX administration. …
- NAID 130004694062
- 長鎖脂肪酸受容体G protein-coupled receptor 40 (GPR40) を介した docosahexaenoic acid (DHA) の抗侵害作用機構
- 中本 賀寿夫,西中 崇,里 尚也 [他],万倉 三正,小山 豊,徳山 尚吾
- 薬学雑誌. 乙号 134(3), 397-403, 2014
- … In addition to intracellular actions, unbound free fatty acids (FFAs) can also carry out extracellular signaling by stimulating the G protein-coupled receptor (GPCR). … Among these receptors, G protein-coupled receptor 40 (GPR40) has been reported to be activated by long-chain fatty acids such as DHA, eicosapentaenoic acid (EPA) and arachidonic acid. …
- NAID 130003391182
Related Links
- Pancreatic Hormone Receptors (n.) 1. (MeSH) Cell surface proteins that bind pancreatic hormones with high affinity and trigger intracellular changes which influence the behavior of cells. These include receptors for glucagon (secreted ...
- Pancreatic Hormone Receptor (n.) 1. (MeSH) Cell surface proteins that bind pancreatic hormones with high affinity and trigger intracellular changes which influence the behavior of cells. These include receptors for glucagon (secreted ...
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★リンクテーブル★
| リンク元 | 「膵ホルモン受容体」 |
| 関連記事 | 「pancreatic」「hormone receptor」「pancreatic hormone」 |
「膵ホルモン受容体」
「pancreatic」
- adj.
- 膵臓の、膵性の、膵の