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1. 大腸癌の分子遺伝学 molecular genetics of colorectal cancer
2. 進行非小細胞肺癌に対する遺伝子型による個別治療 personalized genotype directed therapy for advanced non small cell lung cancer
3. 進行非小細胞肺癌の治療の役割 overview of the treatment of advanced non small cell lung cancer
4. 甲状腺結節および非髄様甲状腺癌におけるオンコジーンおよび腫瘍抑制遺伝子 oncogenes and tumor suppressor genes in thyroid nodules and nonmedullary thyroid cancer
5. 前立腺癌の分子生物学 molecular biology of prostate cancer

English Journal

Effects of high mobility group protein box 1 and toll like receptor 4 pathway on warts caused by human papillomavirus.

Weng H1, Liu H2, Deng Y3, Xie Y4, Shen G2.
Molecular medicine reports.Mol Med Rep.2014 Oct;10(4):1765-71. doi: 10.3892/mmr.2014.2477. Epub 2014 Aug 11.
Accumulative evidence has demonstrated that inflammation has an important role in human papillomavirus (HPV) oncogenicity. However, the effects of high mobility group protein box 1 (HMGB1)-toll like receptor 4 (TLR4) signaling pathway associated inflammation on epidermal warts caused by HPV remain
PMID 25118798

MicroRNA-543 suppresses endometrial cancer oncogenicity via targeting FAK and TWIST1 expression.

Bing L1, Hong C, Li-Xin S, Wei G.
Archives of gynecology and obstetrics.Arch Gynecol Obstet.2014 Sep;290(3):533-41. doi: 10.1007/s00404-014-3219-3. Epub 2014 Apr 4.
INTRODUCTION: Focal adhesion kinase (FAK) is a critical mediator of extracellular matrix signaling, cell survival, proliferation and motility. Twist homolog 1 (TWIST1) is a transcription factor and serves as a powerful oncogene. Increased FAK and TWIST1 expression are observed in a variety of solid
PMID 24699721

Stadler CR1, Vegi N2, Mulaw MA2, Edmaier KE2, Rawat VP2, Dolnik A3, Bullinger L3, Heilmeier B4, Quintanilla-Fend L5, Spiekermann K1, Hiddemann W1, Döhner K3, Döhner H3, Feuring-Buske M3, Buske C2.
Leukemia.Leukemia.2014 Sep;28(9):1838-43. doi: 10.1038/leu.2014.74. Epub 2014 Feb 18.
Although the transforming potential of Hox genes is known for a long time, it is not precisely understood to which extent splicing is important for the leukemogenicity of this gene family. To test this for Hoxa9, we compared the leukemogenic potential of the wild-type Hoxa9, which undergoes natural
PMID 24535405