WordNet
- a state of poor nutrition; can result from insufficient or excessive or unbalanced diet or from inability to absorb foods
- bring disorder to (同)disarray
- a physical condition in which there is a disturbance of normal functioning; "the doctor prescribed some medicine for the disorder"; "everyone gets stomach upsets from time to time" (同)upset
- a disturbance of the peace or of public order
- (physiology) the organic process of nourishing or being nourished; the processes by which an organism assimilates food and uses it for growth and maintenance
- the scientific study of food and drink (especially in humans)
- of or relating to or providing nutrition; "nutritional information" (同)nutritionary
- not arranged in order (同)unordered
PrepTutorEJDIC
- 栄養不良,栄養失調
- 〈U〉『無秩序』,混乱,乱雑(confusion) / 《しばしば複数形で》(社会的・政治的な)粉争,騒動 / 〈C〉(肉体的・精神的な)不調,異常,障害 / …‘の'秩序を乱す / 〈心身〉‘に'異常を起こさせる
- (生物の)栄養摂取 / 栄養分,食物
- 栄養の,栄養的の
UpToDate Contents
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- 1. 食物アレルギーにおける栄養上の問題 nutritional issues in food allergy
- 2. 気晴らし食い-摂食障害：成人における治療の概要 binge eating disorder overview of treatment in adults
- 3. 摂食障害：治療の概要 eating disorders overview of treatment
- 4. Nutritional status in patients with sustained heavy alcohol use
- 5. 重症患者における栄養補給：概要 nutrition support in critically ill patients an overview
English Journal
- Maternal high fat diet consumption during the perinatal period programs offspring behavior.
- Sullivan EL, Nousen EK, Chamlou KA.Author information Department of Biology, University of Portland, Portland, OR, USA; Division of Neuroscience, Oregon National Primate Research Center, Beaverton, OR, USA. Electronic address: sullivae@up.edu.AbstractThe environment that a developing offspring experiences during the perinatal period is markedly influenced by maternal health and diet composition. Evidence from both epidemiological studies and animal models indicates that maternal diet and metabolic status play a critical role in programming the neural circuitry that regulates behavior, resulting in long-term consequences for offspring behavior. Maternal diet and metabolic state influence the behavior of offspring directly by impacting the intrauterine environment and indirectly by modulating maternal behavior. The mechanisms by which maternal diet and metabolic profile shape the perinatal environment remain largely unknown, but recent research has found that increases in inflammatory cytokines, nutrients (glucose and fatty acids), and hormones (insulin and leptin) affect the environment of the developing offspring. Offspring exposed to maternal obesity and high fat diet consumption during development are more susceptible to developing mental health and behavioral disorders such as anxiety, depression, attention deficit hyperactivity disorder, and autism spectrum disorders. Recent evidence suggests that this increased risk for behavioral disorders is driven by modifications in the development of neural pathways involved in behavioral regulation. In particular, research indicates that the development of the serotonergic system is impacted by exposure to maternal obesity and high fat diet consumption, and this disruption may underlie many of the behavioral disturbances observed in these offspring. Given the high rates of obesity and high fat diet consumption in pregnant women, it is vital to examine the influence that maternal nutrition and metabolic profile have on the developing offspring.
- Physiology & behavior.Physiol Behav.2014 Jan 17;123:236-42. doi: 10.1016/j.physbeh.2012.07.014. Epub 2012 Oct 17.
- The environment that a developing offspring experiences during the perinatal period is markedly influenced by maternal health and diet composition. Evidence from both epidemiological studies and animal models indicates that maternal diet and metabolic status play a critical role in programming the n
- PMID 23085399
- The future is now: early life events preset adult behaviour.
- Patchev AV, Rodrigues AJ, Sousa N, Spengler D, Almeida OF.Author information Max Planck Institute of Psychiatry, Munich, Germany.AbstractTo consider the evidence that human and animal behaviours are epigenetically programmed by lifetime experiences. Extensive PubMed searches were carried out to gain a broad view of the topic, in particular from the perspective of human psychopathologies such as mood and anxiety disorders. The selected literature cited is complemented by previously unpublished data from the authors' laboratories. Evidence that physiological and behavioural functions are particularly sensitive to the programming effects of environmental factors such as stress and nutrition during early life, and perhaps at later stages of life, is reviewed and extended. Definition of stimulus- and function-specific critical periods of programmability together with deeper understanding of the molecular basis of epigenetic regulation will deliver greater appreciation of the full potential of the brain's plasticity while providing evidence-based social, psychological and pharmacological interventions to promote lifetime well-being.
- Acta physiologica (Oxford, England).Acta Physiol (Oxf).2014 Jan;210(1):46-57. doi: 10.1111/apha.12140. Epub 2013 Jul 13.
- To consider the evidence that human and animal behaviours are epigenetically programmed by lifetime experiences. Extensive PubMed searches were carried out to gain a broad view of the topic, in particular from the perspective of human psychopathologies such as mood and anxiety disorders. The selecte
- PMID 23790203
- The microvasculature: a target for nutritional programming and later risk of cardio-metabolic disease.
- Musa MG, Torrens C, Clough GF.Author information Vascular Research Group, Human Development and Health, Faculty of Medicine, University of Southampton, Southampton, UK.AbstractThere is compelling evidence that microvascular deficits affecting multiple tissues and organs play an important role in the aetiopathogenesis of cardio-metabolic disease. Furthermore, both in humans and animal models, deficits in small vessel structure and function can be detected early, often before the onset of macrovascular disease and the development of end-organ damage that is common to hypertension and obesity-associated clinical disorders. This article considers the growing evidence for the negative impact of an adverse maternal diet on the long-term health of her child, and how this can result in a disadvantageous vascular phenotype that extends to the microvascular bed. We describe how structural and functional modifications in the offspring microcirculation during development may represent an important and additional risk determinant to increase susceptibility to the development of cardio-metabolic disease in adult life and consider the cell-signalling pathways associated with endothelial dysfunction that may be 'primed' by the maternal environment. Published studies were identified that reported outcomes related to the microcirculation, endothelium, maternal diet and vascular programming using NCBI PubMed.gov, MEDLINE and ISI Web of Science databases from 1980 until April 2013 using pre-specified search terms. Information extracted from over 230 original reports and review articles was critically evaluated by the authors for inclusion in this review.
- Acta physiologica (Oxford, England).Acta Physiol (Oxf).2014 Jan;210(1):31-45. doi: 10.1111/apha.12131. Epub 2013 Jun 25.
- There is compelling evidence that microvascular deficits affecting multiple tissues and organs play an important role in the aetiopathogenesis of cardio-metabolic disease. Furthermore, both in humans and animal models, deficits in small vessel structure and function can be detected early, often befo
- PMID 23758932
Japanese Journal
- Possibility of Polyunsaturated Fatty Acids for the Prevention and Treatment of Neuropsychiatric Illnesses
- Hashimoto Michio,Maekawa Motoko,Katakura Masanori,Hamazaki Kei,Matsuoka Yutaka
- Journal of Pharmacological Sciences, 2014
- … Numerous epidemiological studies have shown that deficits in the dietary PUFA docosahexaenoic acid and eicosapentaenoic acid are associated with the onset and progression of neuropsychiatric illnesses such as dementia, schizophrenia, depression, and posttraumatic stress disorder (PTSD). …
- NAID 130003391498
- Wernicke Encephalopathyの臨床的問題(<特集>ビタミンB研究委員会 平成24年度シンポジウム「B群ビタミンによる疾患の治療」)
- 栗山 勝
- ビタミン 87(11), 617-620, 2013-11-25
- … The new problems of nutritional, medical and social backgrounds have to be studied. … According to autopsy-based studies, the disorder is still underdiagnosed. … Even diagnosing the disorder, an insufficient treatment is followed by mental sequelae such as Korsakoff syndrome. …
- NAID 110009685717
- 神経性食欲不振症患者への在宅中心静脈栄養法の導入の試み(摂食障害の治療の進歩,2012年,第53回日本心身医学会総会ならびに学術講演会(鹿児島))
- 鈴木(堀田) 眞理,荒木 まり子,浦野 綾子 [他],大和田 里奈,市原 敦弘
- 心身医学 53(9), 841-848, 2013-09-01
- 日本の医療行政は入院期間の短縮化と在宅医療を推進している.重症の低体重,電解質異常,腎不全で入院を繰り返さざるを得ない神経性食欲不振症(anorexia nervosa:AN)患者に在宅中心静脈栄養法(home parenteral nutrition:HPN)を導入した.本人と家族にインフォームド・コンセントを行い,訪問看護サービス,在宅支援薬局,地域の医療機関と連携して,HPNの円滑な実施と合 …
- NAID 110009635262
Related Links
- Nutritional diseases are diseases in humans that are directly or indirectly caused by a lack of essential nutrients in the diet. Nutritional diseases are commonly associated with chronic malnutrition. Additionally, conditions such as obesity from ...
- Nutrition disorders can be caused by an insufficient intake of food or of certain nutrients, by an inability of the body to absorb and use nutrients, or by overconsumption of certain foods. Examples include obesity caused by excess energy intake, ...
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★リンクテーブル★
| リンク元 | 「栄養障害」「nutrition disorder」 |
| 関連記事 | 「disorder」「nutritional」 |
「栄養障害」
- 英
- nutritional disorder, malnutrition, nutritional deficiency, malnutritior
- 関
- 栄養失調、栄養不良、栄養不足、栄養失調症、栄養性疾患
「nutrition disorder」
「disorder」
- 障害：個人的苦痛や機能の障害があるので「疾病」とは言えるものの、その背景にある臓器障害がもう一つはっきりしない場合に用いられる。(PSY.9)
- n.
- 注意
- vt.
- 乱す、乱雑にする。(人)の(心身の)調子を狂わせる。
- vi.
「nutritional」
- 栄養性の、栄養上の、栄養的な
- 関
- nutrient、nutritionally、trophic、-trophic