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1. 急性骨髄性白血病の病因 pathogenesis of acute myeloid leukemia
2. 急性骨髄性白血病の臨床症状、病理学的特徴、および診断 clinical manifestations pathologic features and diagnosis of acute myeloid leukemia
3. 急性骨髄性白血病の分子遺伝学 molecular genetics of acute myeloid leukemia
4. 小児および思春期における急性リンパ芽球性白血病の症状および診断の概要 overview of the presentation and diagnosis of acute lymphoblastic leukemia in children and adolescents
5. 慢性骨髄性白血病の細胞生物学および分子生物学 cellular and molecular biology of chronic myeloid leukemia

Maki K, Sugita F, Nakamura Y, Sasaki K, Mitani K.SourceDepartment of Hematology and Oncology, Dokkyo Medical University School of Medicine, 880 Kitakobayashi, Mibu-machi, Shimotsuga-gun, Tochigi, 321-0293, Japan.
International journal of hematology.Int J Hematol.2013 Jan;97(1):83-91. doi: 10.1007/s12185-012-1232-5. Epub 2012 Dec 19.
RUNX1-EVI1 generated by t(3;21) is a causative gene in the leukemic transformation of chronic hematopoietic stem cell tumors. Recruitment of histone deacetylase via carboxyl terminal-binding protein by RUNX1-EVI1 results in transcriptional dysregulation in target genes of wild-type RUNX1, leading to
PMID 23250858

Opposite regulation of vitamin D receptor by ATRA in AML cells susceptible and resistant to vitamin D-induced differentiation.

Gocek E, Marchwicka A, Baurska H, Chrobak A, Marcinkowska E.SourceDepartment of Biotechnology, University of Wroclaw, Tamka 2, 50-137 Wroclaw, Poland.
The Journal of steroid biochemistry and molecular biology.J Steroid Biochem Mol Biol.2012 Nov;132(3-5):220-6. doi: 10.1016/j.jsbmb.2012.07.001. Epub 2012 Jul 10.
Some leukemic cell lines can be driven to differentiate to monocyte-like cells by 1,25-dihydroxyvitamin D(3) (1,25D) and to granulocyte-like cells by all-trans retinoic acid (ATRA). Acute myloid leukemias (AMLs) are heterogeneous blood malignancies characterized by a block at various stages of hemat
PMID 22789609