WordNet

a disorder (usually of unknown origin) of the heart muscle (myocardium) (同)myocardiopathy

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1. 虚血性心筋症の診断およびマネージメント diagnosis and management of ischemic cardiomyopathy
2. 心不全の疫学および原因 epidemiology and causes of heart failure
3. 拡張型心筋症の原因 causes of dilated cardiomyopathy
4. 心不全および心筋症における心臓突然死の一次予防 primary prevention of sudden cardiac death in heart failure and cardiomyopathy
5. 心筋症の心エコーによる認識 echocardiographic recognition of cardiomyopathies

Champion S1, Belcour D2, Vandroux D2, Drouet D2, Gaüzère BA2, Bouchet B2, Bossard G2, Djouhri S2, Jabot J2, Champion M3, Lefort Y2.
European heart journal. Acute cardiovascular care.Eur Heart J Acute Cardiovasc Care.2014 Sep 8. pii: 2048872614547686. [Epub ahead of print]
BACKGROUND: Stress cardiomyopathy (SC) is a transient ventricular dysfunction rarely described in the critical care setting.OBJECTIVE: To evaluate the mechanisms, incidence, treatment and prognosis of SC.METHOD: This is a retrospective observational study of every critically-ill patient admitted to
PMID 25202025

Ca2+ /calmodulin-dependent protein kinase II equally induces sarcoplasmic reticulum Ca2+ leak in human ischaemic and dilated cardiomyopathy.

Fischer TH1, Eiringhaus J, Dybkova N, Förster A, Herting J, Kleinwächter A, Ljubojevic S, Schmitto JD, Streckfuß-Bömeke K, Renner A, Gummert J, Hasenfuss G, Maier LS, Sossalla S.
European journal of heart failure.Eur J Heart Fail.2014 Sep 8. doi: 10.1002/ejhf.163. [Epub ahead of print]
AIMS: The sarcoplasmic reticulum (SR) Ca2+ leak is an important pathomechanism in heart failure (HF). It has been suggested that Ca2+ /calmodulin-dependent protein kinase II (CaMKII) is only relevant for the induction of the SR Ca2+ leak in non-ischaemic but not in ischaemic HF. Therefore, we invest
PMID 25201344

Exendin-4 Improves Cardiac Function in Mice Overexpressing Monocyte Chemoattractant Protein-1 in Cardiomyocytes.

Younce CW1, Niu J2, Ayala J1, Burmeister MA1, Smith LH1, Kolattukudy P2, Ayala JE3.
Journal of molecular and cellular cardiology.J Mol Cell Cardiol.2014 Sep 5. pii: S0022-2828(14)00279-X. doi: 10.1016/j.yjmcc.2014.08.022. [Epub ahead of print]
The incretin hormone glucagon-like peptide-1 (Glp1) is cardioprotective in models of ischemia-reperfusion injury, myocardial infarction and gluco/lipotoxicity. Inflammation is a factor in these models, yet it is unknown whether Glp1 receptor (Glp1r) agonists are protective against cardiac inflammati
PMID 25200599

Crossing the bends: Support-catheter based left ventricular lead placement in challenging cardiac resynchronization therapy device implantation

Kumar Malik Amit,Ching Chi Keong,Tan Boon Yew,Chong Daniel,Teo Wee Siong
Journal of Arrhythmia 28(4), 225-227, 2012
… We describe the case of a 32-year-old male with non-ischaemic cardiomyopathy, left bundle-branch block, and an LV ejection fraction of 30%, who was referred to our centre for a repeat attempt at an LV lead implant. …
NAID 130002129482