WordNet

relating to a technique in which the body is entered by puncture or incision
marked by a tendency to spread especially into healthy tissue; "invasive cancer cells"
malignant tumor originating in glandular epithelium (同)glandular cancer, glandular_carcinoma

PrepTutorEJDIC

(戦争などが)侵略的な / (言葉などが)立ち入った,出過ぎた

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1. 肺悪性腫瘍の病理 pathology of lung malignancies
2. 原発不明の腺癌 adenocarcinoma of unknown primary site
3. 浸潤子宮頸部腺癌 invasive cervical adenocarcinoma
4. 子宮頸部上皮内腺癌 cervical adenocarcinoma in situ
5. 細気管支肺胞上皮癌（上皮内腺癌） bronchioloalveolar carcinoma including adenocarcinoma in situ

English Journal

Differences in the prognosis of resected lung adenocarcinoma according to the histological subtype: a retrospective analysis of Japanese lung cancer registry data.

Sakurai H, Asamura H, Miyaoka E, Yoshino I, Fujii Y, Nakanishi Y, Eguchi K, Mori M, Sawabata N, Okumura M, Yokoi K; Japanese Joint Committee of Lung Cancer Registry.Author information Division of Thoracic Surgery, National Cancer Center Hospital, Tokyo, Japan.AbstractOBJECTIVES: This study intended to assess the clinicopathological features of the histological subtypes of adenocarcinoma of the lung in a large registry population.
European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.Eur J Cardiothorac Surg.2014 Jan;45(1):100-7. doi: 10.1093/ejcts/ezt284. Epub 2013 May 31.
OBJECTIVES: This study intended to assess the clinicopathological features of the histological subtypes of adenocarcinoma of the lung in a large registry population.METHODS: The Japanese Joint Committee of Lung Cancer Registry performed a nationwide retrospective registry study on the prognosis and
PMID 23729748

DCLK1 Marks a Morphologically Distinct Subpopulation of Cells With Stem Cell Properties in Preinvasive Pancreatic Cancer.

Bailey JM1, Alsina J2, Rasheed ZA3, McAllister FM3, Fu YY4, Plentz R5, Zhang H6, Pasricha PJ4, Bardeesy N7, Matsui W3, Maitra A8, Leach SD9.Author information 1Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, Maryland; The McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland.2Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, Maryland.3Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, Maryland.4Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland.5Department of Medicine, Massachusetts General Hospital Cancer Center, Harvard Medical School, Boston, Massachusetts; Department of Internal Medicine, Medical University Hospital, Tuebingen, Germany.6Department of Molecular Microbiology and Immunology, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland.7Department of Medicine, Massachusetts General Hospital Cancer Center, Harvard Medical School, Boston, Massachusetts.8Department of Pathology, The Sol Goldman Pancreatic Cancer Research Center, Johns Hopkins University School of Medicine, Baltimore, Maryland.9Department of Surgery, Johns Hopkins University School of Medicine, Baltimore, Maryland; The McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland. Electronic address: stleach@jhmi.edu.AbstractBACKGROUND & AIMS: As in other tumor types, progression of pancreatic cancer may require a functionally unique population of cancer stem cells. Although such cells have been identified in many invasive cancers, it is not clear whether they emerge during early or late stages of tumorigenesis. Using mouse models and human pancreatic cancer cell lines, we investigated whether preinvasive pancreatic neoplasia contains a subpopulation of cells with distinct morphologies and cancer stem cell-like properties.
Gastroenterology.Gastroenterology.2014 Jan;146(1):245-56. doi: 10.1053/j.gastro.2013.09.050. Epub 2013 Oct 2.
BACKGROUND & AIMS: As in other tumor types, progression of pancreatic cancer may require a functionally unique population of cancer stem cells. Although such cells have been identified in many invasive cancers, it is not clear whether they emerge during early or late stages of tumorigenesis. Usi
PMID 24096005

Identification and manipulation of biliary metaplasia in pancreatic tumors.

Delgiorno KE1, Hall JC2, Takeuchi KK3, Pan FC4, Halbrook CJ2, Washington MK5, Olive KP6, R Spence J7, Sipos B8, Wright CV4, Wells JM9, Crawford HC10.Author information 1Department of Molecular Genetics and Microbiology, Stony Brook University, Stony Brook, New York; Department of Cancer Biology, Mayo Clinic, Jacksonville, Florida.2Department of Cancer Biology, Mayo Clinic, Jacksonville, Florida; Department of Pharmacological Sciences, Stony Brook University, Stony Brook, New York.3Department of Cancer Biology, Mayo Clinic, Jacksonville, Florida.4Department of Cell and Developmental Biology, Vanderbilt University Medical Center, Nashville, Tennessee.5Department of Pathology, Vanderbilt University Medical Center, Nashville, Tennessee.6Departments of Medicine and Pathology, Herbert Irving Comprehensive Cancer Center, Columbia University Medical Center, New York, New York.7Department of Internal Medicine, University of Michigan, Ann Arbor, Michigan.8Department of Pathology, University Hospital Tubingen, Tubingen, Germany.9Department of Developmental Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.10Department of Molecular Genetics and Microbiology, Stony Brook University, Stony Brook, New York; Department of Cancer Biology, Mayo Clinic, Jacksonville, Florida. Electronic address: crawford.howard@mayo.edu.AbstractBACKGROUND & AIMS: Metaplasias often have characteristics of developmentally related tissues. Pancreatic metaplastic ducts are usually associated with pancreatitis and pancreatic ductal adenocarcinoma. The tuft cell is a chemosensory cell that responds to signals in the extracellular environment via effector molecules. Commonly found in the biliary tract, tuft cells are absent from normal murine pancreas. Using the aberrant appearance of tuft cells as an indicator, we tested if pancreatic metaplasia represents transdifferentiation to a biliary phenotype and what effect this has on pancreatic tumorigenesis.
Gastroenterology.Gastroenterology.2014 Jan;146(1):233-244.e5. doi: 10.1053/j.gastro.2013.08.053. Epub 2013 Aug 30.
BACKGROUND & AIMS: Metaplasias often have characteristics of developmentally related tissues. Pancreatic metaplastic ducts are usually associated with pancreatitis and pancreatic ductal adenocarcinoma. The tuft cell is a chemosensory cell that responds to signals in the extracellular environment
PMID 23999170