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1. 腹膜透析における腹膜炎の病態生理および予防 pathophysiology and prevention of peritonitis in peritoneal dialysis
2. ロドコッカス・エクイ感染症の微生物学、病因、および疫学 microbiology pathogenesis and epidemiology of rhodococcus equi infections
3. 腹膜透析における腹膜炎の臨床症状および診断 clinical manifestations and diagnosis of peritonitis in peritoneal dialysis
4. 結核の免疫学 immunology of tuberculosis
5. 肝硬変患者における腹水の病因 pathogenesis of ascites in patients with cirrhosis

Epigenetic Modulation in Periodontitis: Interaction of Adiponectin and JMJD3-IRF4 Axis in Macrophages.

Xuan D1,2, Han Q1,2, Tu Q2, Zhang L2, Yu L2, Murry D2, Tu T2, Tang Y2, Lian JB3, Stein GS3, Valverde P4, Zhang J1, Chen J2.
Journal of cellular physiology.J Cell Physiol.2016 May;231(5):1090-6. doi: 10.1002/jcp.25201. Epub 2015 Oct 6.
Emerging evidence suggests an important role for epigenetic mechanisms in modulating signals during macrophage polarization and inflammation. JMJD3, a JmjC family histone demethylase necessary for M2 polarization is also required for effective induction of multiple M1 genes by lipopolysaccharide (LP
PMID 26399931

Gene expression profile in the activation of subperitoneal fibroblasts reflects prognosis of patients with colon cancer.

Yokota M1,2, Kojima M3, Higuchi Y4, Nishizawa Y1, Kobayashi A1, Ito M1, Saito N1, Ochiai A3.
International journal of cancer. Journal international du cancer.Int J Cancer.2016 Mar 15;138(6):1422-31. doi: 10.1002/ijc.29851. Epub 2015 Oct 1.
Tumors can create a heterogenetic tumor microenvironment. We recently identified the pathologically unique cancer microenvironment formed by peritoneal invasion (CMPI), and revealed that subperitoneal fibroblasts (SPFs) within peritoneal tissue play a crucial role in tumor progression through their
PMID 26370611

Susceptibility and resistance to Echinococcus granulosus infection: Associations between mouse strains and early peritoneal immune responses.

Mourglia-Ettlin G1, Merlino A2, Capurro R3, Dematteis S4.
Immunobiology.Immunobiology.2016 Mar;221(3):418-26. doi: 10.1016/j.imbio.2015.11.012. Epub 2015 Dec 2.
In helminth infections, there are no easy associations between host susceptibility and immune responses. Interestingly, immunity to cestodes - unlike most helminths - seems to require Th1-type effectors. In this sense, we reported recently that Balb/c and C57Bl/6 mice are high and low susceptible st
PMID 26658113