象牙質異形成症、象牙質異形成
WordNet
- an appreciable consequence (especially a lessening); "it made a dent in my bank account"
- an impression in a surface (as made by a blow) (同)ding, gouge, nick
- abnormal development (of organs or cells) or an abnormal structure resulting from such growth
PrepTutorEJDIC
- くぼみ,へこみ / …‘を'くぼませる,へこませる / へこむ,くぼむ
- (くし・歯車などの)歯
Wikipedia preview
出典(authority):フリー百科事典『ウィキペディア(Wikipedia)』「2015/05/24 13:51:46」(JST)
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Dentin dysplasia |
Preoperative panoramic radiographs showing features of dentin dysplasia type I
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Classification and external resources |
ICD-10 |
K00.5 |
ICD-9 |
520.5 |
Teeth appearance in a patient with dentin dysplasia type I
Dentin dysplasia is a genetic disorder of teeth, commonly exhibiting an autosomal dominant inheritance. It is characterized by presence of normal enamel but atypical dentin with abnormal pulpal morphology. There are two types. Type I is the radicular type, and type II is the coronal type. In the radicular type, the roots of teeth are shorter than normal and the pulp chamber may be nearly gone. The pulp chamber is sometimes described as having a "crescent shaped" appearance. In the coronal type, the pulps are enlarged and are described as having a "thistle tube" appearance, in permanent dentition. In the deciduous dentition, coronal dentin dysplasia bears a resemblance to Dentinogenesis Imperfecta type II.
Radiographic Features
Type 1: Roots are short, blunt and conical. In deciduous teeth, pulp chambers and root canals are completely obliterated while in permanent they may be crescent shaped.
Type 2: The pulp chamber of the deciduous teeth become completely obliterated. The permanent teeth displays large pulp chamber in the coronal portion of the tooth - referred to as thistle tube appearance. Pulp stones may be found.
Histologic Features
Type 1: Normal dentinal tubule formation is blocked and new dentin forms around obstacles...known as lava flowing around boulders.
References
Developmental tooth disease/tooth abnormality (K00–K01, 520)
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Quantity |
- Anodontia/Hypodontia
- Hyperdontia
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Shape and size |
- Concrescence
- Fusion
- Gemination
- Dens evaginatus/Talon cusp
- Dens invaginatus
- Enamel pearl
- Macrodontia
- Microdontia
- Taurodontism
- Supernumerary roots
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Formation |
- Dilaceration
- Regional odontodysplasia
- Turner's hypoplasia
- Enamel hypoplasia
- Ectopic enamel
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Other hereditary |
- Amelogenesis imperfecta
- Dentinogenesis imperfecta
- Dentin dysplasia
- Regional odontodysplasia
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Other |
- Dental fluorosis
- Tooth impaction
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Index of teeth
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Description |
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Disease |
- Developmental
- Jaw
- Neoplasms and cancer
- Injury
- Other
- Symptoms and signs
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Treatment |
- Dentistry
- endodontology
- orthodontics
- prosthodontology
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UpToDate Contents
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- 1. 歯の発達障害 developmental defects of the teeth
- 2. 小児における歯周病:関連する全身疾患 periodontal disease in children associated systemic conditions
English Journal
- The fine tuning role of microRNA-RNA interaction in odontoblast differentiation and disease.
- Sun Q1, Liu H, Chen Z.Author information 1State Key Laboratory Breeding Base of Basic Science of Stomatology (Hubei-MOST) and Key Laboratory for Oral Biomedicine of Ministry of Education (KLOBM), School and Hospital of Stomatology, Wuhan University, Wuhan, China.AbstractDentinogenesis imperfecta and dentin dysplasia are two common types of genetic oral diseases resulted from the aberrant differentiation of odontoblast. Understanding the mechanisms of odontoblast differentiation is crucial for finding the diagnosis candidate genes and treatment targets for such kinds of diseases. Previous work has identified a battery of transcription factors and growth factors regulating odontoblast differentiation; however, the post-transcriptional regulating mechanisms of them are poorly studied. MicroRNAs (miRNA) are a group of non-coding RNAs widely studied in organ development, inflammation, and tumorigenesis because of its inhibitory effects on the target mRNAs. Also, miRNAs along with their binding targets form a complex competing endogenous RNA (ceRNA) network where miRNAs serve as the fine tuning balancers between their targets. Recent reports demonstrated the essential role of the miRNA pathway in dentinogenesis and the regulatory role of several specific miRNAs in the in vitro model of odontoblast differentiation. Herein, we will discuss the general roles of miRNA in diseases, the function of miRNAs during odontoblast differentiation, and finally the potential pathological mechanisms through which miRNAs cause the odontoblast-related diseases.
- Oral diseases.Oral Dis.2014 Mar 22. doi: 10.1111/odi.12237. [Epub ahead of print]
- Dentinogenesis imperfecta and dentin dysplasia are two common types of genetic oral diseases resulted from the aberrant differentiation of odontoblast. Understanding the mechanisms of odontoblast differentiation is crucial for finding the diagnosis candidate genes and treatment targets for such kind
- PMID 24654877
- Prevalence of dental anomalies among 7- to 35-year-old people in Hamadan, Iran in 2012-2013 as observed using panoramic radiographs.
- Shokri A1, Poorolajal J2, Khajeh S1, Faramarzi F3, Kahnamoui HM3.Author information 1Department of Oral and Maxillofacial Radiology, School of Dentistry, Hamadan University of Medical Sciences, Hamadan, Iran.2Research Center for Modeling of Noncommunicable Diseases, Department of Epidemiology and Biostatistics, School of Public Health, Hamadan University of Medical Sciences, Hamadan, Iran.3Department of Endodontic, School of Dentistry, Hamadan University of Medical Sciences, Hamadan, Iran.AbstractPURPOSE: This study was performed to evaluate the prevalence of all types and subtypes of dental anomalies among 7- to 35-year-old patients by using panoramic radiographs.
- Imaging science in dentistry.Imaging Sci Dent.2014 Mar;44(1):7-13. doi: 10.5624/isd.2014.44.1.7. Epub 2014 Mar 19.
- PURPOSE: This study was performed to evaluate the prevalence of all types and subtypes of dental anomalies among 7- to 35-year-old patients by using panoramic radiographs.MATERIALS AND METHODS: This cross-sectional study was conducted on 1649 people in Hamadan City, in 2012-2013. The prevalence of f
- PMID 24701453
- Ion channels, channelopathies, and tooth formation.
- Duan X.Author information Department of Oral Biology, Clinic of Oral Rare Diseases and Genetic Diseases, School of Stomatology, The Fourth Military Medical University, Xi'an, 710032, P.R. China.AbstractThe biological functions of ion channels in tooth development vary according to the nature of their gating, the species of ions passing through those gates, the number of gates, localization of channels, tissue expressing the channel, and interactions between cells and microenvironment. Ion channels feature unique and specific ion flux in ameloblasts, odontoblasts, and other tooth-specific cell lineages. Both enamel and dentin have active chemical systems orchestrating a variety of ion exchanges and demineralization and remineralization processes in a stage-dependent manner. An important role for ion channels is to regulate and maintain the calcium and pH homeostasis that are critical for proper enamel and dentin biomineralization. Specific functions of chloride channels, TRPVs, calcium channels, potassium channels, and solute carrier superfamily members in tooth formation have been gradually clarified in recent years. Mutations in these ion channels or transporters often result in disastrous changes in tooth development. The channelopathies of tooth include altered eruption (CLCN7, KCNJ2, TRPV3), root dysplasia (CLCN7, KCNJ2), amelogenesis imperfecta (KCNJ1, CFTR, AE2, CACNA1C, GJA1), dentin dysplasia (CLCN5), small teeth (CACNA1C, GJA1), tooth agenesis (CLCN7), and other impairments. The mechanisms leading to tooth channelopathies are primarily related to pH regulation, calcium homeostasis, or other alterations of the niche for tooth eruption and development.
- Journal of dental research.J Dent Res.2014 Feb;93(2):117-25. doi: 10.1177/0022034513507066. Epub 2013 Sep 27.
- The biological functions of ion channels in tooth development vary according to the nature of their gating, the species of ions passing through those gates, the number of gates, localization of channels, tissue expressing the channel, and interactions between cells and microenvironment. Ion channels
- PMID 24076519
Japanese Journal
- Dandy-Walker variantの患児に歯の形成不全症の合併が疑われた1例
- 増田 啓次,中村 志保,山口 登,小笠原 貴子,山座 治義,西垣 奏一郎,柳田 憲一,廣藤 雄太,野中 和明
- 小児歯科学雑誌 52(3), 440-447, 2014
- Dandy-Walker variant はDandy-Walker malformation の軽症型で,小脳虫部の発生異常と第四脳室の嚢胞状拡張を主な特徴とする。我々はDandy-Walker variant の患児に歯の形成不全症の合併が疑われた1 例を経験した。患児は初診時年齢10 歳5 か月の男児で,歯の実質欠損を伴わない6の根尖性歯周炎およびこれと関連する右側下顎骨周囲膿瘍の精査加療を …
- NAID 130005084436
- Segmental odontomaxillary dysplasia: report of a case emphasizing histopathological, immunohistochemical and scanning electron microscopic features
- S. Azevedo Rebeca,J. da Silveira Luísa,Flávio M. Moliterno Luis,Maria M. A. Miranda Águida,P. de Almeida Oslei,R. Pires Fábio
- Journal of Oral Science 55(3), 259-262, 2013
- … Segmental odontomaxillary dysplasia (SOD) is a rare developmental disorder of the maxilla, and there is little information on its morphological features. … Several dental abnormalities were present, including numerous dentin and pulp defects, altered composition of hard tissue, and proliferation of myofibroblasts in the pulp and the soft tissue surrounding affected teeth. …
- NAID 130003366683
- Dentinogenesis and Dentin Sialophosphoprotein (DSPP)
- YAMAKOSHI Yasuo
- Journal of oral biosciences 51(3), 134-142, 2009-08-20
- NAID 10027104715
Related Links
- A rare case of hereditary disturbance of dentine, Dentin dysplasia type I is presented, which is characterized by short or total absence of roots, obliterated pulp chambers, and peri-apical radiolucencies. It affects both primary and ...
- Summary Dentin dysplasia (DD) is a rare disorder belonging to the group of hereditary dentin defects (see this term) and is characterized by abnormal dentin structure and root development resulting in abnormal tooth development. It ...
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