心房収縮

WordNet

(physiology) a shortening or tensing of a part or organ (especially of a muscle or muscle fiber) (同)muscular_contraction, muscle_contraction
the act of decreasing (something) in size or volume or quantity or scope
a word formed from two or more words by omitting or combining some sounds; "`wont is a contraction of `will not"; "`oclock is a contraction of `of the clock"
of or relating to a cavity or chamber in the body (especially one of the upper chambers of the heart)

PrepTutorEJDIC

〈U〉〈C〉(…の)収縮,短縮《+『of』+『名』》 / 〈U〉(通貨・資金・支出などの)縮小,節減《+『of』+『名』》 / 〈U〉(いやなこと・よくないことを)身に負うこと,(悪習などが)身につくこと《+『of』+『名』》 / 〈C〉陣痛

UpToDate Contents

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1. 発作性心房細動 paroxysmal atrial fibrillation
2. 心房細動および洞調律への心臓除細動の血行動態的影響 hemodynamic consequences of atrial fibrillation and cardioversion to sinus rhythm
3. 心耳の心エコー評価 echocardiographic evaluation of the atria and appendages
4. 心房粗動の概要 overview of atrial flutter
5. 小児における、心房中隔欠損（ASD）の分類および孤立性ASDの臨床的特徴および診断 classification of atrial septal defects asds and clinical features and diagnosis of isolated asds in children

English Journal

Coronary flow dynamics in children after repair of Tetralogy of Fallot.

Aburawi EH1, Munkhammar P2, Carlsson M3, El-Sadig M4, Pesonen E2.Author information 1Departments of Pediatrics, College of Medicine and Health Sciences, United Arab Emirates University, United Arab Emirates; Departments of Pediatric Cardiology, Skåne University Hospital, Lund University, Sweden. Electronic address: e.aburawi@uaeu.ac.ae.2Departments of Pediatric Cardiology, Skåne University Hospital, Lund University, Sweden.3Clinical Physiology and Nuclear Medicine, Skåne University Hospital, Lund University, Sweden.4Institute of Public Health, College of Medicine and Health Sciences, United Arab Emirates University, United Arab Emirates.AbstractOBJECTIVES: To assess the possible effect of a stiff right ventricle on the coronary flow (CF) in patients with post-operative Tetralogy of Fallot (TOF).
International journal of cardiology.Int J Cardiol.2014 Mar 1;172(1):122-6. doi: 10.1016/j.ijcard.2013.12.188. Epub 2014 Jan 9.
OBJECTIVES: To assess the possible effect of a stiff right ventricle on the coronary flow (CF) in patients with post-operative Tetralogy of Fallot (TOF).BACKGROUND: Right ventricular restrictive physiology i.e. forward flow during atrial contraction (RVRP), is characteristic to many patients with po
PMID 24462136

β3-Adrenergic Regulation of L-Type Ca2+ Current and Force of Contraction in Human Ventricle.

Treinys R, Zablockaitė D, Gendvilienė V, Jurevičius J, Skeberdis VA.Author information Institute of Cardiology, Lithuanian University of Health Sciences, 17 Sukilėlių Avenue, 50009, Kaunas, Lithuania.Abstractβ3-Adrenergic receptor (β3-AR) is expressed in human atrial and ventricular tissues. Recently, we have demonstrated that it was involved in the activation of L-type Ca2+ current (I Ca,L) in human atrial myocytes and the force of contraction of human atrial trabeculae. In the present study, we examined the effect of β3-AR agonist CGP12177 which also is a β1-AR/β2-AR antagonist on I Ca,L in human ventricular myocytes (HVMs) and the force of contraction of human ventricular trabeculae. CGP12177 stimulated I Ca,L in HVMs with high potency but much lower efficacy than isoprenaline. The β3-AR antagonist L-748,337 inhibited the effect of CGP12177. CGP12177 and L748,337 competed selectively on β3-ARs because L748,337 had no effect on isoprenaline-induced stimulation of I Ca,L, while CGP12177 completely blocked the effect of isoprenaline. The activation of β3-ARs by CGP12177 does not involve the activation of Gi proteins because CGP12177 had no effect on forskolin-induced stimulation of I Ca,L. CGP12177 had no effect on the force of contraction of human ventricular trabeculae. L-NMMA, an inhibitor of NO synthase, and IBMX, a nonselective inhibitor of phosphodiesterases, did not potentiate the effect of CGP12177 either on contraction of human ventricular trabeculae or on I Ca,L in HVMs. We conclude that in human ventricles β3-AR activation has no inotropic effect, while it slightly increases I Ca,L. In contrast to human atrium, the activation of β3-ARs in human ventricle is not accompanied by increased activity of phosphodiesterases.
The Journal of membrane biology.J Membr Biol.2014 Feb 15. [Epub ahead of print]
β3-Adrenergic receptor (β3-AR) is expressed in human atrial and ventricular tissues. Recently, we have demonstrated that it was involved in the activation of L-type Ca2+ current (I Ca,L) in human atrial myocytes and the force of contraction of human atrial trabeculae. In the present study, we exam
PMID 24531741

Proven cardiac changes during death-feigning (tonic immobility) in rabbits (Oryctolagus cuniculus).

Giannico AT, Lima L, Lange RR, Froes TR, Montiani-Ferreira F.Author information Department of Veterinary Medicine, Federal University of Paraná, Rua dos Funcionários, 1540, Curitiba, PR, 80035-050, Brazil, amaliaturner@uol.com.br.AbstractTonic immobility (TI) is a response to fear or threat by remaining motionless, principally when attacked by predators from which there is no possibility of escape. Thus, here we demonstrate a way of easily reproducing this phenomenon in a laboratory setting and characterize the cardiac electromechanical alterations during TI. We observed a significant decrease in heart rate (HR) and changes of rhythm in electrocardiogram during TI in rabbits. Echocardiogram showed a significant increase in the left ventricle chamber diameter during systole and a consequent decrease in fractional shortening and ejection fraction, in addition to the HR and rhythm changes. There was also a significant decrease in aortic and pulmonary artery blood flow. Diastolic functional changes included a significant decrease of the peak atrial contraction velocity (A peak) and consequent increase in the ratio of peak early diastolic velocity to A peak and increased isovolumetric relaxation time. We were able to prove that TI changes the cardiac function considerably. Although the "fight-or-flight" response is the most common response to fear, which is characterized by the action of sympathetic nervous system with tachycardia and increased physical activity, TI is an alternative anti-predator behavior causing cardiac changes opposite to the "fight-or-flight" phenomenon.
Journal of comparative physiology. A, Neuroethology, sensory, neural, and behavioral physiology.J Comp Physiol A Neuroethol Sens Neural Behav Physiol.2014 Feb 11. [Epub ahead of print]
Tonic immobility (TI) is a response to fear or threat by remaining motionless, principally when attacked by predators from which there is no possibility of escape. Thus, here we demonstrate a way of easily reproducing this phenomenon in a laboratory setting and characterize the cardiac electromechan
PMID 24515628