出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2014/12/27 16:14:20」(JST)[Wiki en表示]
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- 1. 過換気症候群 hyperventilation syndrome
- 2. 頸動脈内膜剥離術および頸動脈ステント留置術の麻酔 anesthesia for carotid endarterectomy and carotid stenting
- 3. 成人における頭蓋内圧亢進の評価およびマネージメント evaluation and management of elevated intracranial pressure in adults
- 4. 小児における頭蓋内圧亢進（ICP） elevated intracranial pressure icp in children
- 5. 動悸を呈する小児に対するアプローチ approach to the child with palpitations
- Association Between Hypocapnia and Febrile Seizures.
- Kilicaslan B1, Erol I, Ozkale Y, Saygi S, Sariturk C.Author information 11Baskent University Faculty of Medicine, Adana Teaching and Medical Research Center, Department of Pediatrics, Adana, Turkey.AbstractThe purpose of this study is to determine whether hyperthermia-induced hyperventilation with subsequent hypocapnia is relevant to febrile seizures in children. This is only the second study to measure pCO2 and pH values in children with febrile seizures. This prospective case-control study enrolled 18 children who presented with febrile seizures and 18 children who presented with a febrile illness without seizures. Venous blood gas analyses were measured both from the febrile seizure and control group. There was no significant difference in mean blood pH between the febrile seizure and control groups but blood pCO2 was significantly lower in the febrile seizure group. Patients with complex febrile seizures exhibited significantly lower pCO2 levels within 1 hour of seizure onset than patients with simplex febrile seizures. These data indicate that febrile seizures may be associated with hyperventilation and that the ensuing hypocapnia may contribute to the development of febrile seizures.
- Journal of child neurology.J Child Neurol.2014 May;29(5):599-602. Epub 2014 Jan 5.
- The purpose of this study is to determine whether hyperthermia-induced hyperventilation with subsequent hypocapnia is relevant to febrile seizures in children. This is only the second study to measure pCO2 and pH values in children with febrile seizures. This prospective case-control study enrolled
- PMID 24396127
- Adolescent girls with emotional disorders have a lower end-tidal CO2 and increased respiratory rate compared with healthy controls.
- Henje Blom E1, Serlachius E, Chesney MA, Olsson EM.Author information 1Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden; Osher Center for Integrative Medicine, Karolinska Institutet, Stockholm, Sweden; Department of Psychiatry, University of California San Francisco, San Francisco, USA.AbstractHyperventilation has been linked to emotional distress in adults. This study investigates end-tidal carbon dioxide (ETCO2 ), respiratory rate (RR), and heart rate variability (HRV) in adolescent girls with emotional disorders and healthy controls. ETCO2 , RR, HRV, and ratings of emotional symptom severity were collected in adolescent female psychiatric patients with emotional disorders (n = 63) and healthy controls (n = 62). ETCO2 and RR differed significantly between patients and controls. ETCO2 , HR, and HRV were significant independent predictors of group status, that is, clinical or healthy, while RR was not. ETCO2 and RR were significantly related to emotional symptom severity and to HRV in the total group. ETCO2 and RR were not affected by use of selective serotonin reuptake inhibitors. It is concluded that emotional dysregulation is related to hyperventilation in adolescent girls. Respiratory-based treatments may be relevant to investigate in future research.
- Psychophysiology.Psychophysiology.2014 May;51(5):412-8. doi: 10.1111/psyp.12188. Epub 2014 Feb 27.
- Hyperventilation has been linked to emotional distress in adults. This study investigates end-tidal carbon dioxide (ETCO2 ), respiratory rate (RR), and heart rate variability (HRV) in adolescent girls with emotional disorders and healthy controls. ETCO2 , RR, HRV, and ratings of emotional symptom se
- PMID 24571123
- The effect of centrally injected CDP-choline on respiratory system; involvement of phospholipase to thromboxane signaling pathway.
- Topuz BB1, Altinbas B1, Yilmaz MS2, Saha S3, Batten TF3, Savci V2, Yalcin M4.Author information 1Department of Physiology, Faculty of Veterinary Medicine, Uludag University, Bursa 16059, Turkey.2Department of Pharmacology and Clinical Pharmacology, Faculty of Medicine, Uludag University, Bursa 16059, Turkey.3Division of Cardiovascular and Diabetes Research, Leeds Institute of Genetics, Health and Therapeutics, University of Leeds, Leeds LS2 9JT, UK.4Department of Physiology, Faculty of Veterinary Medicine, Uludag University, Bursa 16059, Turkey. Electronic address: firstname.lastname@example.org.AbstractCDP-choline is an endogenous metabolite in phosphatidylcholine biosynthesis. Exogenous administration of CDP-choline has been shown to affect brain metabolism and to exhibit cardiovascular, neuroendocrine neuroprotective actions. On the other hand, little is known regarding its respiratory actions and/or central mechanism of its respiratory effect. Therefore the current study was designed to investigate the possible effects of centrally injected CDP-choline on respiratory system and the mediation of the central cholinergic receptors and phospholipase to thromboxane signaling pathway on CDP-choline-induced respiratory effects in anaesthetized rats. Intracerebroventricularly (i.c.v.) administration of CDP-choline induced dose- and time-dependent increased respiratory rates, tidal volume and minute ventilation of male anaesthetized Spraque Dawley rats. İ.c.v. pretreatment with atropine failed to alter the hyperventilation responses to CDP-choline whereas mecamylamine, cholinergic nicotinic receptor antagonist, mepacrine, phospholipase A2 inhibitor, and neomycin phospholipase C inhibitor, blocked completely the hyperventilation induced by CDP-choline. In addition, central pretreatment with furegrelate, thromboxane A2 synthesis inhibitor, also partially blocked CDP-choline-evoked hyperventilation effects. These data show that centrally administered CDP-choline induces hyperventilation which is mediated by activation of central nicotinic receptors and phospholipase to thromboxane signaling pathway.
- Respiratory physiology & neurobiology.Respir Physiol Neurobiol.2014 May 1;195:50-8. doi: 10.1016/j.resp.2014.02.005. Epub 2014 Feb 18.
- CDP-choline is an endogenous metabolite in phosphatidylcholine biosynthesis. Exogenous administration of CDP-choline has been shown to affect brain metabolism and to exhibit cardiovascular, neuroendocrine neuroprotective actions. On the other hand, little is known regarding its respiratory actions a
- PMID 24560778
- 野村 広徳,洪 陽子,金城 孝則,野々村 大地,米田 傑,山本 致之,鄭 則秀,高田 晋吾,松宮 清美
- 泌尿器科紀要 59(8), 535-538, 2013-08
- … Tetany due to hyperventilation syndrome appeared on postoperative day42. … After re-removal of the urethral catheter, however, hyperventilation syndrome recurred. …
- NAID 120005322751
- 過換気症候群 (特集 呼吸器救急 対応のコツとピットフォール回避法) -- (救急疾患・病態)
- 齋藤 兄治
- 救急医学 = The Japanese journal of acute medicine 37(6), 689-693, 2013-06
- NAID 40019704558
- Low Serum Concentrations of Vitamin B6 and Iron Are Related to Panic Attack and Hyperventilation Attack
- Mikawa Yasuhito,Mizobuchi Satoshi,Egi Moritoki,Morita Kiyoshi
- Acta Medica Okayama 67(2), 99-104, 2013-04
- … Patients undergoing a panic attack (PA) or a hyperventilation attack (HVA) are sometimes admitted to emergency departments (EDs). …
- NAID 120005232500
- Hyperventilation occurs when the rate and quantity of alveolar ventilation of carbon dioxide exceeds body's production of carbon dioxide.   Hyperventilation can be voluntary or involuntary. When alveolar ventilation is excessive ...
- Hyperventilation Information Including Symptoms, Diagnosis, Treatment, Causes, Videos, Forums, and local community support. Find answers to health issues you can trust from BetterMedicine.com ... Introduction What is ...