高アルミニウム血症
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- 1. 肝臓の合成能の検査(アルブミン、凝固因子、プロトロンビン時間など)tests of the livers biosynthetic capacity eg albumin coagulation factors prothrombin time [show details]
…curve. Acute hyperalbuminemia is seen most commonly in patients with volume depletion , in whom it is often associated with hemoconcentration as well. A case report described hyperalbuminemia in a patient …
- 2. 総血清カルシウムとイオン化血清カルシウム濃度の相関relation between total and ionized serum calcium concentrations [show details]
…presence of a normal ionized calcium concentration can occur in patients with hypoalbuminemia, hyperalbuminemia, and multiple myeloma. If the total serum calcium is low but the ionized calcium is normal, …
- 3. 高カルシウム血症への診断的アプローチdiagnostic approach to hypercalcemia [show details]
…authorities prefer to measure the serum ionized calcium in this situation. In patients with hyperalbuminemia (eg, due to severe dehydration), serum albumin is elevated, and there may be an associated elevation …
- 4. 成人における代謝性アシドーシスへのアプローチapproach to the adult with metabolic acidosis [show details]
…generate an anion gap metabolic acidosis. This is due to hypoperfusion-induced lactic acidosis, hyperalbuminemia (albumin is negatively charged and accounts for most of the anion gap in normal individuals)…
- 5. 高カルシウム血症の治療treatment of hypercalcemia [show details]
… concentration may be normal when serum ionized calcium is elevated. Alternatively, patients with hyperalbuminemia due to severe volume depletion and rare patients with multiple myeloma, who have a calcium-binding …
English Journal
Japanese Journal
- 2, 2'-ジヒドロキシァゾベンゼンを用いる吸光検出-イオン対逆相分配高速液体クロマトグラフィによる血清アルミニウムと鉄の同時測定法
- 郡司 法民,佐藤 松男,金子 恵美子,星野 仁,四ッ柳 隆夫
- 臨床化学 21(2), 113-118, 1992
- われわれは慢性血液透析患者の金属の体内動態を知るため, 血清中のアルミニウムと鉄の同時測定法を検討し, 高速液体クロマトグラフィを用いる高感度で選択性の高い方法を開発した。方法は, 血清を酸加水分解および除タンパクすることで遊離したアルミニウムと鉄を, 2, 2'-ジヒドロキシァゾベンゼンを用いてキレートとし, このキレ-ト反応液100μlを注入し, ODSカラムを用いてイオン対逆相分配モードでア …
- NAID 130003357977
- 維持透析患者における高アルミニウム血症およびそれに伴う諸症状に対するデスフェリオキサミンの長期少量投与法の有効性と安全性
- 倉持 元
- 日本腎臓学会誌 31(3), 267-273, 1989
- … In order to examine the efficacy and safety of long-term and low-dose desferrioxamine (DFO) therapy against hyperaluminemia and the clinical symptoms associated with hyperaluminemia, 4 patients (3 men and 1 woman, 40-62 years old, period of hemodialysis : 69-189 months) undergoing maintenance hemodialysis were treated by DFO (0.5 g/week) and hemodiafiltration for 27 weeks. …
- NAID 130004169574
- 梅田 優,海本 浩一,泉 暢英,上水流 雅人,西尾 正一,前川 正信
- 人工臓器 17(1), 98-101, 1988
- フレームレス原子吸光光度計をもちいて、健常者10例(1群)、透析をうけていないがアルミニウム(Al)製剤を投与されていない慢性腎炎患者27例(II群), CAPD患者7例(III群), HD患者30例(IV群)の血清Al (s-Al)及び赤血球内Al (RBC-Al)を測定した。HD患者ではs-Alとアルミゲル®の投与量とは正相関を示したが、RBG-Alは投与量と相関がなく、非投与群でも高 …
- NAID 130001734593
Related Links
- High Albumin (Hyperalbuminemia): Symptoms, Causes, Treatment. The High albumin Is medically known as hyperalbuminemia, and means an excess or high concentration of albumin in the blood. The serum albumin concentration is typically 35-50 g / L (3.5-5.0 g / dl). Proteins are basic components of the human body, so albumin and globulin are essential ...
- A 12-year-old, neutered male, mixed-breed dog was presented to The Ohio State University Veterinary Teaching Hospital with a history of weight loss and weakness. Laboratory abnormalities reported by the referring veterinarian during
- Research of Hyperalbuminemia has been linked to Proteinuria Of Undiagnosed Cause, Azotemia, Dehydration, Hyperphosphatemia, Cat Diseases. The study of Hyperalbuminemia has been mentioned in research publications which can be found using our bioinformatics tool below. Researched pathways related to Hyperalbuminemia include Excretion, Transport ...
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- 英
- hyperaluminemia
- 正常人ではあまり問題にならないとされている。
- アルミニウムが排泄している病態、例えば、腎機能が低下している例では、医原性あるいは経口的にアルミニウムが供給されると体内に蓄積し、アルミニウム血症を呈する。
参考
- 1. Water content of aluminum, dialysis dementia, and osteomalacia.
- Wills MR, Savory J.
- Environmental health perspectives.Environ Health Perspect.1985 Nov;63:141-7.
- In the presence of normal renal function, a high concentration of aluminum in drinking water has been implicated as a factor in the etiology of a neurological syndrome in one specific geographical area. The role of aluminum as a toxic agent in other neurological disorders, where renal function is normal, is controversial. Aluminum is absorbed from the gastrointestinal tract and is normally excreted by the kidneys in the urine. In patients with chronic renal failure, aluminum appears to be of proven toxicological importance. In these patients the accumulation of aluminum in tissues causes an encephalopathy (dialysis encephalopathy or dialysis dementia), a specific form of metabolic bone disease (osteomalacic dialysis osteodystrophy), and an anemia and also plays an etiological role in some of the other complications associated with end-stage chronic renal disease. A failure in the normal renal excretory mechanism accounts for the tissue accumulation in chronic renal failure. The majority of chronic renal failure patients who develop aluminum toxicity are on long-term treatment with either hemo- or peritoneal dialysis; some patients develop toxicity who are only on treatment with aluminum-containing phosphate-binding agents. Aluminum in the dialysate appears to be the major source of the metal in chronic renal failure patients who develop aluminum toxicity. The aluminum content of the dialysate depends primarily on the content of the water with which it is prepared; there may be some contribution from the chemicals used in the concentrate which is added to the water. Some domestic tap-water supplies contain aluminum in high concentration, either naturally or because aluminum has been added as a flocculant in the purification process.(ABSTRACT TRUNCATED AT 250 WORDS)
- PMID 3908086