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1. カルシウムおよびリン酸バランスの調節 regulation of calcium and phosphate balance
2. 高リン血症の原因および治療の概要 overview of the causes and treatment of hyperphosphatemia
3. グルコース-6-リン酸脱水素酵素欠損症の診断および治療 diagnosis and treatment of glucose 6 phosphate dehydrogenase deficiency
4. 慢性腎疾患における高リン血症の治療 treatment of hyperphosphatemia in chronic kidney disease
5. 低リン血症の評価および治療 evaluation and treatment of hypophosphatemia

[Reduction of the immune responses and synthesis of cytokines associated with Th1 and Th2 lymphocyte function and their correction by polyoxidonium under conditions of subacute intoxication by organophosphorus compounds].

Zabrodskiĭ PF, Kirichuk VF, Iafarova IKh, Lim VG, Svistunov AA.
Eksperimental'naia i klinicheskaia farmakologiia.Eksp Klin Farmakol.2009 Nov-Dec;72(6):33-5.
Experiments on Wistar rats showed that subacute poisoning by organophosphorus compounds dimethyldichlorovinyl phosphate (DDVF), malation, and dimethylparation (total dose, 1.0 LD50) suppresses both cell and humoral immune responses and significantly decreases the level of blood cytokines (IFNg, IL-4
PMID 20095398

Inhibition of function of T cell subpopulations and decrease in cytokine production during subacute poisoning with various toxicants.

Zabrodskii PF1, Germanchuk VG, Mandych VG.
Bulletin of experimental biology and medicine.Bull Exp Biol Med.2008 Aug;146(2):234-6.
Experiments on outbred albino rats showed that subacute intoxication with organophosphorus compounds dimethyldichlorovinyl phosphate and malathion primarily decreased functional activity of Th1 lymphocytes, immune reactions associated with these cells, and interferon-g production compared to that of
PMID 19145326

[Effect T-activin on the activity of the natural killer cells after acute intoxication by toxic chemical substances].

Zabrodskiĭ PF1, Germanchuk VG, Kirichuk VF, Molotkov AO, Mal'tseva GM, Osipov OV, Nodel' ML.
Eksperimental'naia i klinicheskaia farmakologiia.Eksp Klin Farmakol.2003 May-Jun;66(3):47-9.
The results of experiments on male mongrel mice showed that a three-day treatment with T-activin in a dose of 2.5 micrograms/kg restored the activity of natural killer cells reduced by acute poisoning (1 LD50) with ethylene glycol (EG), methanol (MeOH), and ethanol (EtOH). In a dose of 5 micrograms/
PMID 12924234