WordNet

a substance that exerts some force or effect
a businessman who buys or sells for another in exchange for a commission (同)factor, broker
any agent or representative of a federal agency or bureau (同)federal agent
a representative who acts on behalf of other persons or organizations
an active and efficient cause; capable of producing a certain effect; "their research uncovered new disease agents"
an encapsulated neural structure consisting of a collection of cell bodies or neurons
the act of obstructing or deflecting someones movements (同)block

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1. 成人における迅速導入気管挿管のための神経筋遮断剤（NMBA） neuromuscular blocking agents nmba for rapid sequence intubation in adults
2. 重症患者における神経筋遮断剤の使用 use of neuromuscular blocking medications in critically ill patients
3. パーキンソン病の病因および発症機序 etiology and pathogenesis of parkinson disease
4. 小児における後天性末梢性ニューロパチーの概要 overview of acquired peripheral neuropathies in children
5. 神経筋接合部に作用する毒素の概要 overview of neuromuscular junction toxins

Voltage-gated Ca2+ influx and mitochondrial Ca2+ initiate secretion from Aplysia neuroendocrine cells.

Hickey CM1, Groten CJ, Sham L, Carter CJ, Magoski NS.
Neuroscience.Neuroscience.2013 Oct 10;250:755-72. doi: 10.1016/j.neuroscience.2013.07.023. Epub 2013 Jul 19.
Neuroendocrine secretion often requires prolonged voltage-gated Ca(2+) entry; however, the ability of Ca(2+) from intracellular stores, such as endoplasmic reticulum or mitochondria, to elicit secretion is less clear. We examined this using the bag cell neurons, which trigger ovulation in Aplysia by
PMID 23876326

Neurokinins inhibit low threshold inactivating K+ currents in capsaicin responsive DRG neurons.

Sculptoreanu A1, Artim DE, de Groat WC.
Experimental neurology.Exp Neurol.2009 Oct;219(2):562-73. doi: 10.1016/j.expneurol.2009.07.016. Epub 2009 Jul 23.
Neurokinins (NK) released from terminals of dorsal root ganglion (DRG) neurons may control firing of these neurons by an autofeedback mechanism. In this study we used patch clamp recording techniques to determine if NKs alter excitability of rat L4-S3 DRG neurons by modulating K(+) currents. In caps
PMID 19631644

Regulation of calcitonin gene-related peptide secretion from trigeminal nerve cells by botulinum toxin type A: implications for migraine therapy.

Durham PL1, Cady R, Cady R.
Headache.Headache.2004 Jan;44(1):35-42; discussion 42-3.
OBJECTIVES: To determine the effect of botulinum toxin type A on calcitonin gene-related peptide secretion from cultured trigeminal ganglia neurons.BACKGROUND: The ability of botulinum toxins to cause muscle paralysis by blocking acetylcholine release at the neuromuscular junction is well known. Pre
PMID 14979881