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- 1. 思春期におけるカルシウムの所要量 calcium requirements in adolescents
- 2. 骨粗鬆症におけるカルシウムおよびビタミンDの補充 calcium and vitamin d supplementation in osteoporosis
- 3. カルシウムおよびリン酸バランスの調節 regulation of calcium and phosphate balance
- 4. 成人におけるカルシウム結石の危険因子 risk factors for calcium stones in adults
- 5. 成人における再発性カルシウム結石の予防 prevention of recurrent calcium stones in adults
- Sources of circulating 3,5,3'-triiodothyronine in hyperthyroidism estimated after blocking of type 1 and type 2 iodothyronine deiodinases.
- Laurberg P1, Vestergaard H, Nielsen S, Christensen SE, Seefeldt T, Helleberg K, Pedersen KM.Author information 1Department of Endocrinology and Medicine, Aalborg Hospital, Aarhus University Hospital, Aalborg, Denmark. email@example.comAbstractCONTEXT: Graves' hyperthyroidism and multinodular toxic goiter lead to high serum T(3) compared with serum T(4). The source of this high T(3) has not been clarified.
- The Journal of clinical endocrinology and metabolism.J Clin Endocrinol Metab.2007 Jun;92(6):2149-56. Epub 2007 Mar 27.
- CONTEXT: Graves' hyperthyroidism and multinodular toxic goiter lead to high serum T(3) compared with serum T(4). The source of this high T(3) has not been clarified.OBJECTIVE: Our objective was to assess the role of iodothyronine deiodinase type 1 (D1) and type 2 (D2) for T(3) production and to esti
- PMID 17389703
- Hashimoto's thyroiditis and the role of selenium. Current concepts.
- Mazokopakis EE1, Chatzipavlidou V.Author information 1Department of Internal Medicine, Naval Hospital of Crete, Chania, Greece. firstname.lastname@example.orgAbstractHashimoto's thyroiditis (HT) is part of the spectrum of autoimmune thyroid diseases. Clinical manifestations of HT are variable and commonly include diffuse or nodular goiter with euthyroidism, subclinical hypothyroidism and permanent hypothyroidism. Uncommonly, HT causes acute destruction of thyroid tissue and release of stored thyroid hormones, causing transient thyrotoxicosis (hashitoxicosis). The contribution of methods and techniques of nuclear medicine to diagnosis and differential diagnosis of HT is indisputable. In HT patients with overt hypothyroidism L-thyroxine (L-T(4)) should be given in the usual replacement doses, but in HT patients with a large goiter and normal or elevated serum thyroid-stimulating hormone (TSH), L-T(4) may be given in doses sufficient to suppress serum TSH. Symptomatic patients with hashitoxicosis and low 24-hour thyroid radioactive iodine ((123)I or (123)I) uptake (RIU) may be treated with beta-blockers (as propranolol) and sodium ipodate or iopanoic acid (iodinated contrast agents) that block the peripheral conversion of T(4) to T(3). Recent clinical studies have documented the suppressive effect of selenium treatment on serum anti-thyroid peroxidase concentrations in patients with HT.
- Hellenic journal of nuclear medicine.Hell J Nucl Med.2007 Jan-Apr;10(1):6-8.
- Hashimoto's thyroiditis (HT) is part of the spectrum of autoimmune thyroid diseases. Clinical manifestations of HT are variable and commonly include diffuse or nodular goiter with euthyroidism, subclinical hypothyroidism and permanent hypothyroidism. Uncommonly, HT causes acute destruction of thyroi
- PMID 17450242
- AuthorsCheng KT.
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