- 関
- anoxic-ischemic encephalopathy、cerebral hypoxia-ischemia、cerebral ischemia-hypoxia、hypoxic-ischemic encephalopathy
WordNet
- kill by smashing someones skull
- mental ability; "hes got plenty of brains but no common sense" (同)brainpower, learning_ability, mental capacity, mentality, wit
- that part of the central nervous system that includes all the higher nervous centers; enclosed within the skull; continuous with the spinal cord (同)encephalon
- the brain of certain animals used as meat
- hit on the head
- local anemia in a given body part sometimes resulting from vasoconstriction or thrombosis or embolism (同)ischaemia
- oxygen deficiency causing a very strong drive to correct the deficiency
PrepTutorEJDIC
- 『脳』,脳髄 / 《しばしば複数形で》『頭脳』,『知力』 / 《話》秀才,知的指導者 / …‘の'頭を打ち砕く
- =brassiere, brassiere
UpToDate Contents
全文を閲覧するには購読必要です。 To read the full text you will need to subscribe.
- 1. 脳卒中評価の概要 overview of the evaluation of stroke
- 2. 脳卒中の病因、分類、および疫学 etiology classification and epidemiology of stroke
- 3. 成人における急性肝不全：マネージメントおよび予後 acute liver failure in adults management and prognosis
- 4. 動脈瘤性くも膜下出血の治療 treatment of aneurysmal subarachnoid hemorrhage
- 5. 脳室周囲白質軟化症 periventricular leukomalacia
English Journal
- Resident microglia, rather than blood-derived macrophages, contribute to the earlier and more pronounced inflammatory reaction in the immature compared with the adult hippocampus after hypoxia-ischemia.
- Umekawa T1,2, Osman AM1, Han W1, Ikeda T2, Blomgren K1.
- Glia.Glia.2015 Dec;63(12):2220-30. doi: 10.1002/glia.22887. Epub 2015 Jul 14.
- The mechanisms of neuronal injury after hypoxia-ischemia (HI) are different in the immature and the adult brain, but microglia activation has not been compared. The purpose of this study was to phenotype resident microglia and blood-derived macrophages in the hippocampus after HI in neonatal (postna
- PMID 26179283
- Erythropoietin as a new therapeutic opportunity in brain inflammation and neurodegenerative diseases.
- Merelli A1, Czornyj L2,3, Lazarowski A1,3.
- The International journal of neuroscience.Int J Neurosci.2015 Nov;125(11):793-7. doi: 10.3109/00207454.2014.989321. Epub 2015 Jan 9.
- Highly expressed Erythropoietin Receptor (EPO-R) has been detected in several nonhematopoietic hypoxic cells, including cells from different brain areas in response to many different types of cell injury. In brain, hypoxia-ischemia (HI) can induce a wide spectrum of biologic responses, where inflamm
- PMID 25405533
- Matrix metalloproteinases as therapeutic targets for stroke.
- Yang Y1, Rosenberg GA2.
- Brain research.Brain Res.2015 Oct 14;1623:30-8. doi: 10.1016/j.brainres.2015.04.024. Epub 2015 Apr 25.
- Matrix metalloproteinases (MMPs) are important in injury and recovery in ischemic injury. They are proteolytic enzymes that degrade all components of the extracellular matrix (ECM). They are secreted in a latent form, protecting the cell from damage, but once activated induce injury prior to rapid i
- PMID 25916577
Japanese Journal
- Chronic treatment with a water-soluble extract from the culture medium of Ganoderma lucidum mycelia (MAK) prevents apoptosis and necroptosis in hypoxia/ischemia-induced injury of type 2 diabetic mouse brain
- 玄 美燕,岡﨑 真理,岩田 直洋,浅野 哲,神内 伸也,松崎 広和,坂本 武史,Miyano Yoshiyuki,飯塚 博,日比野 康英
- Evidence-based complementary and alternative medicine 2015(865986), 1-29, 2015-02-03
- … On the basis of our previous study regarding a water-soluble extract from the culture medium of Ganoderma lucidum mycelia (designed as MAK), which exerts antioxidative and neuroprotective effects, the present study was conducted to evaluate the preventive effects of MAK on apoptosis and necroptosis, a programmed necrosis induced by hypoxia/ischemia (H/I) in type 2 diabetic KKAy mice. …
- NAID 120005590299
- 発達期脳病変に続発する持続的筋収縮状態の臨床像
- 丸山 幸一,井合 瑞江,荒井 洋,横地 健治
- 脳と発達 46(1), 10-15, 2014
- 【目的】発達期脳病変による運動障害においてときに観察される, 全身の緊張が長時間持続する状態を「持続的筋収縮状態」と定義し, その臨床像を明らかにする. 【方法】診断基準を満たす66症例を後方視的に調査した. 【結果】早産核黄疸の症例では非対称性姿勢をとり, 姿勢保持や快刺激で症状の緩和を認めた. 他の症例は基底核・視床と大脳の両側広汎性病変が中核であり, 後弓反張位が多く, 姿勢保持や刺激によ …
- NAID 130005005688
- HYPOXIC-ISCHEMIC BRAIN INJURY IN NEWBORN RAT : NEUROPROTECTIVE EFFECTS OF ACETAMINOPHEN AND THE INVOLVEMENT OF ACID SPHINGOMYELINASE ACTIVATION
- Oguma Rena,Ishida-Nakajima Wako,Kawamura Masanari [他],Miura Shinobu,Oyama Chikako,Sato Yoko,Arai Hirokazu,Takahashi Tsutomu
- 秋田医学 39(3-4), 109-118, 2013-03
- … In this study, we used the Rice-Vannucci model to examine whether the administration of acetaminophen was protective against hypoxic-ischemic brain injury in immature rat brain. …
- NAID 110009575398
Related Links
- Brain Hypoxia and Ischemia Gabriel G. Haddad, M.D. and Shan Ping Yu, M.D., Ph.D., Editors Brain Hypoxia and Ischemia explores the various aspects of cell ... Most up-to-date volume to focus on the mechanisms of cell injury and ...
- Hypoxic-ischemic brain injury is a diagnostic term that encompasses a complex constellation of pathophysiological and molecular injuries to the brain induced by hypoxia, ischemia, cytotoxicity, or combinations of these conditions ...
Related Pictures
★リンクテーブル★
| リンク元 | 「低酸素性虚血性脳症」「anoxic-ischemic encephalopathy」「cerebral hypoxia-ischemia」「cerebral ischemia-hypoxia」「脳低酸素虚血」 |
| 関連記事 | 「brain hypoxia」「hypoxia」「ischemia」「brain」 |
「低酸素性虚血性脳症」
- 英
- hypoxic-ischemic encephalopathy hypoxic ischemic encephalopathy, anoxic-ischemic encephalopathy, brain hypoxia-ischemia
- 関
- 低酸素性脳虚血, 脳低酸素虚血, 無酸素性虚血性脳症, 脳虚血低酸素、無酸素性脳症
「anoxic-ischemic encephalopathy」
- 関
- brain hypoxia-ischemia、cerebral hypoxia-ischemia、cerebral ischemia-hypoxia、hypoxic-ischemic encephalopathy
「cerebral hypoxia-ischemia」
- 関
- anoxic-ischemic encephalopathy、brain hypoxia-ischemia、cerebral ischemia-hypoxia、hypoxic-ischemic encephalopathy
「cerebral ischemia-hypoxia」
- 関
- anoxic-ischemic encephalopathy、brain hypoxia-ischemia、cerebral hypoxia-ischemia、hypoxic-ischemic encephalopathy
「脳低酸素虚血」
「brain hypoxia」
- 関
- anoxic encephalopathy、brain anoxia、cerebral anoxia、cerebral hypoxia、hypoxic brain damage、hypoxic encephalopathy
「hypoxia」
- n.
「ischemia」
- n.
「brain」