aspermia

Aspermia
Classification and external resources
Specialty	urology
ICD-10	N46
ICD-9-CM	606.0
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無精液症

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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2016/03/03 06:54:08」(JST)

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-spermia,

Further information: Testicular infertility factors

(A)spermia - complete lack of semen

(Asthenozoo)spermia - reduced sperm motility

(Azoo)spermia - absence of sperm cells in semen

(Hyper)spermia - large semen volume

(Hypo)spermia - small semen volume

(Oligozoo)spermia - few spermatozoa in semen

(Necrozoo)spermia - dead or immobile sperms

(Teratozoo)spermia - sperm with abnormal morphology

Aspermia is the complete lack of semen with ejaculation (not to be confused with azoospermia, the lack of sperm cells in the semen). It is associated with infertility.

One of the causes of aspermia is retrograde ejaculation,^[1] which can be brought on by excessive drug use, or as a result of prostate surgery. It can also be caused by alpha blockers such as tamsulosin and silodosin.

Another cause of aspermia is ejaculatory duct obstruction, which may result in a complete lack of or a very low-concentration semen (oligospermia), in which the semen contains only the secretion of accessory prostate glands downstream to the orifice of the ejaculatory ducts.

Aspermia can be caused by androgen deficiency.^[2]^[3]^[4]^[5] This can be the result of absence of puberty, in which the prostate gland and seminal vesicles (which are the main sources of semen) remain small due to lack of androgen exposure and do not produce seminal fluid, or of treatment for prostate cancer, such as maximal androgen blockade.^[6]

References

^ UCSB's SexInfo
^ Eberhard Nieschlag; Hermann Behre (29 June 2013). Andrology: Male Reproductive Health and Dysfunction. Springer Science & Business Media. pp. 54–. ISBN 978-3-662-04491-9.
^ Wayne J.G. Hellstrom (28 November 2012). Androgen Deficiency and Testosterone Replacement: Current Controversies and Strategies. Springer Science & Business Media. pp. 34–. ISBN 978-1-62703-179-0.
^ Carrie Bagatell; William J. Bremner (27 May 2003). Androgens in Health and Disease. Springer Science & Business Media. pp. 314–. ISBN 978-1-59259-388-0.
^ Susan Blackburn (14 April 2014). Maternal, Fetal, & Neonatal Physiology. Elsevier Health Sciences. pp. 39–. ISBN 978-0-323-29296-2.
^ John J. Mulcahy (1 January 2001). Male Sexual Function. Springer Science & Business Media. pp. 3–. ISBN 978-1-59259-098-8.

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English Journal

Oral Toxicity of 2,4-Dinitroanisole in Rats.

Lent EM1, Crouse LC2, Wallace SM2.
International journal of toxicology.Int J Toxicol.2016 Oct 7. pii: 1091581816670321. [Epub ahead of print]
Subacute and subchronic studies were conducted to assess the toxicity of 2,4-dinitroanisole (DNAN) and to provide information important for protecting the health of military and civilian personnel. In the subchronic study, male and female Sprague-Dawley rats were dosed with DNAN via oral gavage at 0
PMID 27765881

Effects of Saikokaryukotsuboreito on Spermatogenesis and Fertility in Aging Male Mice.

Zang ZJ1, Ji SY, Zhang YN, Gao Y, Zhang B.
Chinese medical journal.Chin Med J (Engl).2016 Apr 5;129(7):846-53. doi: 10.4103/0366-6999.178972.
BACKGROUND: Aspermia caused by exogenous testosterone limit its usage in late-onset hypogonadism (LOH) patients desiring fertility. Saikokaryukotsuboreito (SKRBT) is reported to improve serum testosterone and relieve LOH-related symptoms. However, it is unclear whether SKRBT affects fertility. We ai
PMID 26996482

Association of single nucleotide polymorphisms in UBR2 gene with idiopathic aspermia or oligospermia in Sichuan, China.

Ma TJ1,2,3, Zhang XJ4, Ding XP1,2,3, Chen HH1,2,3, Zhang YW1,2,3, Ding M1,2,3.
Andrologia.Andrologia.2016 Mar 4. doi: 10.1111/and.12569. [Epub ahead of print]
The associations between three single nucleotide polymorphisms (SNPs; rs3749897, rs16895863 and rs373341) of UBR2 gene and idiopathic aspermia or oligospermia were investigated in this study by a case-control experiment with 149 fertile and 316 infertile men, including 244 patients with idiopathic a
PMID 26940145