WordNet

a thin pliable sheet of material
a pliable sheet of tissue that covers or lines or connects the organs or cells of animals or plants (同)tissue layer
shaped like a sausage (同)sausage-shaped

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(生物の)膜,皮膜

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1. 血液-血液透析膜の相互作用に関与する生化学的機序 biochemical mechanisms involved in blood hemodialysis membrane interactions
2. 赤血球細胞膜の動態および機構 red blood cell membrane dynamics and organization
3. ライソゾーム膜蛋白質2欠損症（糖原病IIb型、ダノン病） lysosome associated membrane protein 2 deficiency glycogen storage disease iib danon disease
4. 補体系の制御因子および受容体 regulators and receptors of the complement system
5. 血液透析膜の生体適合性についての臨床結果 clinical consequences of hemodialysis membrane biocompatibility

English Journal

Allantoid cyst in the umbilical cord diagnosed with B-flow ultrasound.

Eggebø TM1, Dirdal HU, Vonbrandis P.Author information 1Department of Obstetrics and Gynecology, Stavanger University Hospital, Stavanger, Norway. tme@lyse.netAbstractAllantiod cysts are true cysts in the umbilical cord. They arise from persistent structures of allantois, and the cysts are filled with urine because of a connection to the urinary bladder. Allantoid cysts are located centrally in the umbilical cord and separate the umbilical cord vessels. B-flow ultrasound is a new technique illustrating blood flow. This method is independent of the insonation angle and therefore superior to Doppler ultrasound visualising long segments of vessels with a winding course. The authors present a case with allantoid cysts diagnosed with B-flow ultrasound.
BMJ case reports.BMJ Case Rep.2012 Aug 27;2012. pii: bcr0320126064. doi: 10.1136/bcr.03.2012.6064.
Allantiod cysts are true cysts in the umbilical cord. They arise from persistent structures of allantois, and the cysts are filled with urine because of a connection to the urinary bladder. Allantoid cysts are located centrally in the umbilical cord and separate the umbilical cord vessels. B-flow ul
PMID 22927270

Chicken embryo as an experimental model for the study of gastroschisis.

Gandara CA1, Araújo ES, Motta UI.Author information 1Caxias do Sul, RS, Brazil. gandara@terra.com.brAbstractPURPOSE: To reproduce the experimental model of gastroschisis in chicken embryos and to prove that the histopathological changes that occur in this model can be compared to those in human gastroschisis.
Acta cirúrgica brasileira / Sociedade Brasileira para Desenvolvimento Pesquisa em Cirurgia.Acta Cir Bras.2008 May-Jun;23(3):247-52.
PURPOSE: To reproduce the experimental model of gastroschisis in chicken embryos and to prove that the histopathological changes that occur in this model can be compared to those in human gastroschisis.METHODS: A total of 278 Leghorn hen (Gallus domesticus) eggs were used. The embryos were divided i
PMID 18552995

Inhibition of vascular endothelial growth factor receptor 2-mediated endothelial cell activation by Axl tyrosine kinase receptor.

Gallicchio M1, Mitola S, Valdembri D, Fantozzi R, Varnum B, Avanzi GC, Bussolino F.Author information 1Department of Anatomy, Pharmacology and Forensic Medicine, University of Turin, C. so Massimo D'azeglio 52, 10100 Torino, Italy. margherita.gallicchio@unito.itAbstractGAS6, the product of a growth arrest specific (GAS) gene, is the ligand of the tyrosine kinase receptor Axl. GAS6 and Axl are both expressed in endothelial cells, where they are involved in many processes such as leukocyte transmigration through capillaries and neointima formation in injured vessels. Here, we show that Axl stimulation by GAS6 results in inhibition of the ligand-dependent activation of vascular endothelial growth factor (VEGF) receptor 2 and the consequent activation of an angiogenic program in vascular endothelial cells. GAS6 inhibits chemotaxis of endothelial cells stimulated by VEGF-A isoforms, but not that triggered by fibroblast growth factor-2 or hepatocyte growth factor. Furthermore, it inhibits endothelial cell morphogenesis on Matrigel and VEGF-A-dependent vascularization of chick chorion allantoid membrane. GAS6 activates the tyrosine phosphatase SHP-2 (SH2 domain-containing tyrosine phosphatase 2), which is instrumental in the negative feedback exerted by Axl on VEGF-A activities. A dominant-negative SHP-2 mutant, in which Cys 459 is substituted by Ser, reverted the effect of GAS6 on stimulation of VEGF receptor 2 and endothelial chemotaxis triggered by VEGF-A. These studies provide the first demonstration of a cross talk between Axl and VEGF receptor 2 and add new information on the regulation of VEGF-A activities during tissue vascularization.
Blood.Blood.2005 Mar 1;105(5):1970-6. Epub 2004 Oct 26.
GAS6, the product of a growth arrest specific (GAS) gene, is the ligand of the tyrosine kinase receptor Axl. GAS6 and Axl are both expressed in endothelial cells, where they are involved in many processes such as leukocyte transmigration through capillaries and neointima formation in injured vessels
PMID 15507525