WordNet

fasten or attach with tape; "tape the shipping label to the box"
a recording made on magnetic tape; "the several recordings were combined on a master tape" (同)tape recording, taping
measuring instrument consisting of a narrow strip (cloth or metal) marked in inches or centimeters and used for measuring lengths; "the carpenter should have used his tape measure" (同)tapeline, tape_measure
a long thin piece of cloth or paper as used for binding or fastening; "he used a piece of tape for a belt"; "he wrapped a tape around the package"
the finishing line for a foot race; "he broke the tape in record time"
the 20th letter of the Roman alphabet (同)t

PrepTutorEJDIC

〈U〉(細長い紙・布などの荷造り用)『テープ』,ひも / 〈C〉(決勝線用の)テープ / =tape measure / 〈C〉〈U〉録音用テープ / =tape recording / =tape measure / …‘を'テープをくくる,テープをくっつける / =tape-record / …‘を'巻き尺で測る / 《しばしば受動態で》《米》…‘に'包帯をする(《英》strap) / =tape-record
tritiumの化学記号
tellurium の化学記号
tantalumの化学記号

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1. 脳性麻痺のマネージメントおよび予後 management and prognosis of cerebral palsy
2. 疥癬 scabies
3. 皮膚疣贅 cutaneous warts
4. 女性の腹圧性尿失禁に対する外科的マネージメント：恥骨後式中部尿道スリング術 surgical management of stress urinary incontinence in women retropubic midurethral slings
5. 女性の腹圧性尿失禁に対する外科的マネージメント：経閉鎖孔式中部尿道スリング術 surgical management of stress urinary incontinence in women transobturator midurethral slings

English Journal

A new spontaneous animal model of NIDDM without obesity in the musk shrew.

Ohno T, Yoshida F, Ichikawa Y, Matsuo S, Hotta N, Terada M, Tanaka S, Yamashita K, Namikawa T, Kitoh J.Author information Department of Parasitology, Hamamatsu University School of Medicine, Japan.AbstractThe EDS (early-onset diabetes in suncus) colony has been developed as a new closed breeding colony of the musk shrew (Suncus murinus, Insectivora) exhibiting a high incidence of spontaneous diabetes mellitus. We investigated the characteristic features of diabetic shrews in this colony. All diabetic shrews are characterized by glycosuria (Tes-tape value > or = 3+), hyperglycemia (23.3 +/- 0.8 mmol/l) and polyuria, and they were affected by the age of 3 months. Cumulative incidence (64.1% in males and 27.8% in females) was kept intact after the age of 3 months. The growth pattern of diabetic shrews was similar to that of non-diabetic shrews, and obesity was not consistent in diabetic shrews. The intraperioneal glucose tolerance test revealed both impaired glucose tolerance and impaired insulin secretion in diabetic shrews. Insulin sensitivity of diabetic shrews decreased in the intraperioneal insulin tolerance test. Neither severe hypertrophy nor lymphocytic infiltration was observed in pancreatic islets of diabetic shrews. These facts suggested that diabetic shrews in the EDS colony should be classified as early-onset non-insulin dependent diabetes mellitus (NIDDM) without obesity. Early-onset of severe hyperglycemia with impaired glucose tolerance is a distinctive character compared with other non-obese NIDDM models in rodents. We concluded that the diabetic shrews in the EDS colony are a new animal model of human NIDDM without obesity.
Life sciences.Life Sci.1998;62(11):995-1006.
The EDS (early-onset diabetes in suncus) colony has been developed as a new closed breeding colony of the musk shrew (Suncus murinus, Insectivora) exhibiting a high incidence of spontaneous diabetes mellitus. We investigated the characteristic features of diabetic shrews in this colony. All diabetic
PMID 9515557

Mucosal IgA antibody against Helicobacter pylori in chronic gastritis and intestinal metaplasia detected by the Tes-Tape method in resection specimens after gastrectomy for gastric cancer.

Matsukura N, Onda M, Tokunaga A, Matsuda N, Yamashita K.Author information First Department of Surgery, Nippon Medical School, Tokyo, Japan.AbstractBACKGROUND: Subjects with atrophic body gastritis have a high prevalence of Helicobacter pylori seropositivity and a low prevalence of H. pylori infection. Disappearance of the organism appears to correlate with the development of intestinal metaplasia. To investigate this point, intestinal metaplasia was biochemically subclassified into complete and incomplete types by the Tes-Tape method, and tissue IgA and IgG antibodies against H. pylori were measured by enzyme-linked immunosorbent assay (ELISA).
Cancer.Cancer.1995 Mar 15;75(6 Suppl):1472-7.
BACKGROUND: Subjects with atrophic body gastritis have a high prevalence of Helicobacter pylori seropositivity and a low prevalence of H. pylori infection. Disappearance of the organism appears to correlate with the development of intestinal metaplasia. To investigate this point, intestinal metaplas
PMID 7889477

[Helicobacter pylori in peptic ulcer and gastric cancer].

Matsukura N, Onda M, Yamashita K.Author information First Dept. of Surgery, Nippon Medical School.AbstractRecently many reports have shown a strong association between Helicobacter pylori infection in the stomach and recurrent peptic ulcer. Moreover, prospective cohort serological studies showed that H. pylori infected individuals have significantly increased rate of gastric cancer in the USA. H. pylori is a gram-negative spiral organism which has urease activity and produces ammonia and CO2 from urea, and nestles in the gastric pits and overlaying mucus gel layer. Many diagnostic methods of H. pylori infection are available; ie bacterial culture, 13C-urea breath test, histology, serum IgG antibody against H. pylori. We developed a new method, ie tissue IgA antibody against H. pylori and detection of H. pylori DNA in the gastric juice by PCR method. Triple therapies with metronidazole, bismuth compounds, and amoxicillin or tetracyclin are difficult to use in Japan because of their sever side effects. Thus, new methods with proton pump inhibitor (PPI) and amoxicillin have been introduced. We treated 14 patients of whom were H. pylori positive-active peptic ulcer with 30 mg/day of lansoprazole, a new PPI, plus 1,500 mg/day of amoxicillin for 2 weeks and 8 (57%) patients were eradicated. Gastric carcinogenesis are multi-steps and multifactorials process. Hypothetical sequence of intestinal type of gastric cancer is that superficial gastritis-->atrophic gastritis-->intestinal metaplasia-->dysplasia-->gastric cancer and H. pylori infection may play a role in the early stage of the sequence. We examined mucosal IgA antibody against H. pylori in chronic gastritis and intestinal metaplasia detected by the Tes-Tape method in 25 resected specimens after gastrectomy for gastric cancer. Positivity rates of tissue H. pylori IgA antibody were lower in the mucosa of intestinal metaplasia than in non-metaplastic gastric mucosa and were negative in carcinoma. Causal relationship between H. pylori infection and gastric cancer is not proven and factors other than H. pylori infection are also important in the gastric carcinogenesis. Finally we introduce 2 reports: (1) NIH Consensus Conference: Helicobacter pylori in peptic ulcer disease (JAMA. 1994; 272: 65-69). The consensus panel concluded that 1. ulcer patients with H. pylori infection require treatment with antimicrobial agents in addition to antisecretory drugs whether on first presentation with the illness or on recurrence; 2. the value of treating nonulcerative dyspepsia patients with H. pylori infection remains to be determined; and 3. the interesting relationship between H. pylori infection and gastric cancer requires further exploration. (2) World Health Organization: Working Group Meeting (Reported in World Congress of Gastroenterology, Los Angeles, 1994). H. pylori plays a causal role in the chain of events leading to cancer of the stomach. Group I: definite carcinogen.
Gan to kagaku ryoho. Cancer & chemotherapy.Gan To Kagaku Ryoho.1995 Feb;22(2):169-78.
Recently many reports have shown a strong association between Helicobacter pylori infection in the stomach and recurrent peptic ulcer. Moreover, prospective cohort serological studies showed that H. pylori infected individuals have significantly increased rate of gastric cancer in the USA. H. pylori
PMID 7857088

Japanese Journal

カドミウム腎症のスクリーニング検査としての尿タンパク，尿糖定性・定量試験の検討

日本衛生学雑誌 45(5), 1014-1019, 1990
NAID 130000998172

Ｎ‐メチル‐Ｎ‐ニトロ‐Ｎ‐ニトロソグアニジン（ＭＮＮＧ）による実験的腸上皮化生ならびに胃しゅりゅうの発生と糖質栄養との関連性

日本栄養・食糧学会誌 37(4), 369-372, 1984
NAID 130000861471

胃粘膜の腸上皮化生について

日本消化器外科学会雑誌 16(3), 497-506, 1983-03-01
NAID 110001359938

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