English Journal

Double dissociation between regulation of conditioned disgust and taste avoidance by serotonin availability at the 5-HT(3) receptor in the posterior and anterior insular cortex.

Tuerke KJ1, Limebeer CL, Fletcher PJ, Parker LA.
The Journal of neuroscience : the official journal of the Society for Neuroscience.J Neurosci.2012 Oct 3;32(40):13709-17. doi: 10.1523/JNEUROSCI.2042-12.2012.
A taste associated with emetic drugs produces conditioned disgust reactions in rats (predominantly gaping), unlike nonemetic drugs that can still produce conditioned taste avoidance but not conditioned disgust. That difference suggests nausea is a prerequisite for learning disgust reactions to taste
PMID 23035083

Serotonergic and dopaminergic mechanisms in graft-induced dyskinesia in a rat model of Parkinson's disease.

Shin E1, Garcia J, Winkler C, Björklund A, Carta M.
Neurobiology of disease.Neurobiol Dis.2012 Sep;47(3):393-406. doi: 10.1016/j.nbd.2012.03.038. Epub 2012 Apr 12.
Dyskinesia seen in the off-state, referred as graft-induced dyskinesia (GID), has emerged as a serious complication induced by dopamine (DA) cell transplantation in parkinsonian patients. Although the mechanism underlying the appearance of GID is unknown, in a recent clinical study the partial 5-HT(
PMID 22579773

Neonatal serotonin (5-HT) depletion does not affect spatial learning and memory in rats.

Piechal A1, Blecharz-Klin K, Wyszogrodzka E, Kołomańska P, Rok-Bujko P, Krząścik P, Kostowski W, Widy-Tyszkiewicz E, Filip M, Stefański R.
Pharmacological reports : PR.Pharmacol Rep.2012;64(2):266-74.
BACKGROUND: Extensive previous research has suggested a role for serotonin (5-HT) in learning and memory processes, both in healthy individuals and pathological disorders including depression, autism and schizophrenia, most of which have a developmental onset. Since 5-HT dysfunction in brain develop
PMID 22661175